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1.14.14.80: long-chain fatty acid omega-monooxygenase

This is an abbreviated version!
For detailed information about long-chain fatty acid omega-monooxygenase, go to the full flat file.

Word Map on EC 1.14.14.80

Reaction

a long-chain fatty acid
+
[reduced NADPH-hemoprotein reductase]
 +
O2
=
an omega-hydroxy-long-chain fatty acid
 +
[oxidized NADPH-hemoprotein reductase]
 +
H2O

Synonyms

At1g69500, At2g27690, CYP4A, CYP4A1, CYP4A11, CYP4A14, CYP4A2, CYP4A3, CYP4A8, CYP4V2, CYP52-E3, CYP52-M1, CYP52-N1, CYP52E3, CYP52M1, CYP52N1, CYP704B1, CYP86A, CYP86A1, CYP86A33, CYP94C1, cytochrome P450 4A11, cytochrome P450 704B1, cytochrome P450 86A1, EC 1.14.13.205, Fatty acid omega-hydroxylase, Lauric acid omega-hydroxylase, omega-hydroxylase, P450 4A11

ECTree

     1 Oxidoreductases
         1.14 Acting on paired donors, with incorporation or reduction of molecular oxygen
             1.14.14 With reduced flavin or flavoprotein as one donor, and incorporation of one atom of oxygen into the other donor
                1.14.14.80 long-chain fatty acid omega-monooxygenase

Expression

Expression on EC 1.14.14.80 - long-chain fatty acid omega-monooxygenase

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EXPRESSION
ORGANISM
UNIPROT
LITERATURE
CYP704B1 is highly coexpressed with CYP703A2, a cytochrome P450 involved in exine production
CYP86A1 transcripts are repressed 2.4fold by 27h of iodoacetc acid treatment and5.9fold by 27 h of salicylic acid treatment
CYP86A2 transcripts are transiently induced 2.5fold by 3 h of wounding and abscisic acid treatment, 2.3fold by 3 h of mannitol treatment, 1.7fold by 3 h of iodoacetic acid treatment, 1.6fold by 3 h of clofibrate treatment, more continuously induced 2.0- and 1.9fold by 30 and 120 min of drought treatment, and induced 1.9- to 2.3fold in etiolated and dark-adapted seedlings
CYP86A4 transcripts are induced 1.9fold by long-term treatment with 1-aminocyclopropane-1-carboxylic acid and repressed 9.0fold by short-term treatment with 1-aminocyclopropane-1-carboxylic acid
CYP86A4 transcripts are repressed 9.0fold by short-term treatment with 1-aminocyclopropane-1-carboxylic acid and induced 1.9fold by long-term treatment with 1-aminocyclopropane-1-carboxylic acid. CYP86A4 transcripts are repressed 3.6fold by 3 h of wounding and 2.4fold in etiolated seedlings
CYP86A7 transcripts are significantly repressed by 2 h of drought treatment, 3 h of salicylic acid and wounding treatments, 3 and 27 h of 1-aminocyclopropane-1-carboxylic acid and mannitol treatments, and in both etiolated and dark-adapted seedlings
CYP86A7 transcripts are transiently induced 1.9fold by 3 h of clofibrate treatment, more continuously induced 1.6 and 1.5fold by 3 and 27 h of methyl jasmonate treatment, and slowly induced 1.7fold by 27 h abscisic acid treatment
CYP86A8 transcripts are induced by 3-h iodoacetic acid and abscisic acid treatments
CYP86A8 transcripts are repressed by 30 min of wounding
in ciprofibrate-treated cells, CYP4A expression increases by 1.5- to 1.8fold
-
isoform CYP52M1 is clearly upregulated during sophorolipid synthesis