EC Number |
Disease |
PubMed ID |
Title of Publication |
Category |
Confidence Level |
---|
2.4.1.255 | Adenocarcinoma of Lung |
30123425 |
High O-linked N-acetylglucosamine transferase expression predicts poor survival in patients with early stage lung adenocarcinoma. |
causal interaction diagnostic usage unassigned |
4 4 0 |
2.4.1.255 | Alzheimer Disease |
12911634 |
UDP-N-acetylglucosaminyl transferase (OGT) in brain tissue: temperature sensitivity and subcellular distribution of cytosolic and nuclear enzyme. |
ongoing research unassigned |
2 0 |
2.4.1.255 | Alzheimer Disease |
16714295 |
Ataxin-10 interacts with O-linked beta-N-acetylglucosamine transferase in the brain. |
causal interaction unassigned |
4 0 |
2.4.1.255 | Alzheimer Disease |
25148700 |
The mitochondrial O-linked N-acetylglucosamine transferase (mOGT) in the diabetic patient could be the initial trigger to develop Alzheimer disease. |
unassigned |
0 |
2.4.1.255 | Alzheimer Disease |
27956640 |
Loss of O-GlcNAc glycosylation in forebrain excitatory neurons induces neurodegeneration. |
ongoing research unassigned |
4 0 |
2.4.1.255 | Alzheimer Disease |
29154269 |
Early and Persistent O-GlcNAc Protein Modification in the Streptozotocin Model of Alzheimer's Disease. |
causal interaction ongoing research unassigned |
2 1 0 |
2.4.1.255 | Breast Neoplasms |
30952976 |
O-GlcNAc Transferase Inhibition Differentially Affects Breast Cancer Subtypes. |
causal interaction therapeutic application unassigned |
4 1 0 |
2.4.1.255 | Carcinogenesis |
27331873 |
O-linked N-acetylglucosamine transferase promotes cervical cancer tumorigenesis through human papillomaviruses E6 and E7 oncogenes. |
unassigned |
0 |
2.4.1.255 | Carcinogenesis |
27703839 |
Formal modeling and analysis of the hexosamine biosynthetic pathway: role of O-linked N-acetylglucosamine transferase in oncogenesis and cancer progression. |
therapeutic application unassigned |
1 0 |
2.4.1.255 | Carcinogenesis |
29435130 |
O-linked N-acetylglucosamine transferase enhances secretory clusterin expression via liver X receptors and sterol response element binding protein regulation in cervical cancer. |
causal interaction ongoing research unassigned |
2 1 0 |