EC Number   |
Protein Variants |
Reference |
|---|
   2.4.1.173 | more |
a T-DNA insertion mutant of UGT713B1/At5g24750 shows a phenotype that seems to be indistinguishable from Col-0 wild-type. A homozygous mutant for ugt713B1, mutation in the in the 5' UTR, seems to express mRNA in semi-quantitative RT-PCR experiments. Construction of an ugt80A2,713B1 double mutant which has a similar sterol profile to the ugt80A2 single mutant showing reduced steryl glucoside content |
-, 736570 |
| 2.4.1.173 | more |
analysis of the function of SGTs by virus-induced gene silencing (VIGS) of SGTL1, SGTL2 and SGTL4 in Withania somnifer. Phenotype, overview |
760139 |
| 2.4.1.173 | more |
analysis of the function of SGTs by virus-induced gene silencing (VIGS) of SGTL1, SGTL2 and SGTL4 in Withania somnifera. Phenotype, overview |
760139 |
| 2.4.1.173 | more |
construction of ugt80A2,713B1 and ugt80A2,B1 double mutants which have similar sterol profiles to the ugt80A2 and ugt80B1 single mutants, respectively, showing reduced steryl glucoside content |
-, 736570 |
| 2.4.1.173 | more |
generation of a ugt80A2;B1 double mutant that is more resistant ot infection by Bortrytis cinerea than the wild-type and shows increased levels of jasmonic acid (JA) and upregulation of two marker genes (PDF1.2 and PR4) of the ERF branch of the JA signaling pathway. The mutant also accumulates higher levels of camalexin, the major Arabidopsis thaliana phytoalexin, than wild-type plants. Camalexin accumulation correlates with enhanced transcript levels of several cytochrome P450 camalexin biosynthetic genes, as well as of their transcriptional regulators WRKY33, ANAC042, and MYB51, suggesting that the Botrytis-induced accumulation of camalexin is coordinately regulated at the transcriptional level, Mutant phenotype, overview |
-, 759256 |
| 2.4.1.173 | more |
generation of a ugt80A2;B1 double mutant that is more resistant to infection by Bortrytis cinerea than the wild-type and shows increased levels of jasmonic acid (JA) and upregulation of two marker genes (PDF1.2 and PR4) of the ERF branch of the JA signaling pathway. The mutant also accumulates higher levels of camalexin, the major Arabidopsis thaliana phytoalexin, than wild-type plants. Camalexin accumulation correlates with enhanced transcript levels of several cytochrome P450 camalexin biosynthetic genes, as well as of their transcriptional regulators WRKY33, ANAC042, and MYB51, suggesting that the Botrytis-induced accumulation of camalexin is coordinately regulated at the transcriptional level. Mutant phenotype, overview |
-, 759256 |
| 2.4.1.173 | more |
generation of ugt80B1 mutants. Sterol glucosides patterns of wild-type and mutant roots, overview. The ugt80B1 mutant shows a significant reduction in stigmasteryl glucosides only. Root hair patterning and GL2 expression are aberrant in ugt80B1 mutants. Expression of upstream cell fate regulators, SCM and WER, is altered in ugt80B1 mutants. Phenotype, overview. The ugt80B1 mutant phenotype is complemented with pro35S:UGT80B1:GFP or with proUGT80B1:UGT80B1:GFP as observed by rescue of the transparent testa phenotype |
-, 760156 |
| 2.4.1.173 | more |
homozygous mutants for ugt80B1, mutation at the exon-intron boundary following the second exon, is a mRNA knockdown allel. Construction of ugt80A2,B1 double mutant which has a similar sterol profiles to the ugt80A2 and ugt80B1 single mutants, respectively, showing reduced steryl glucoside content |
-, 736570 |
| 2.4.1.173 | more |
mutation of isoform UGT80B1 principally alters embryonic development and seed suberin accumulation and cutin formation in the seed coat, leading to abnormal permeability and tetrazolium salt uptake, mutations in UGT80A2 and USG80B1 genes result in reduced seed size, transparent testa, and salt uptake phenotypes |
706323 |
| 2.4.1.173 | more |
null mutant by deletion of ugt52 gene with no enzyme activity |
637784 |
