EC Number   |
Inhibitors |
Structure  |
|---|
    1.1.1.284 | more |
it is unclear whether activation or inhibition by fatty acids is of physiological importance |
 |
| 1.1.1.284 | more |
not inhibited by 0.4 mM 2-aminodecanoic acid and 10 mM glutathionesulfonic acid |
|
| 1.1.1.284 | more |
GSNOR inhibitors may be novel tools for regulating nitric oxide bioactivity and assessing the role of S-nitrosothiols in vivo |
|
| 1.1.1.284 | more |
strong oxidizing agents are capable of reducing Arabidopsis thaliana GSNOR activity. Plant systems reversibly inhibit their GSNOR activity in response to oxidative radicals |
|
| 1.1.1.284 | more |
plant systems reversibly inhibit their GSNOR activity in response to oxidative radicals |
|
| 1.1.1.284 | more |
oxidative post-translationally modification of GSNOR inhibits the activity of the enzyme suggesting a direct crosstalk between reactive oxygen species (ROS)- and reactive nitrogen species (RNS)-signaling |
|
| 1.1.1.284 | more |
GSNOR enzymatic activity, but not gene expression, is inhibited by the nitrogen assimilatory pathway via post-transcriptional S-nitrosation, preventing any scavenging of GSNO |
|
| 1.1.1.284 | more |
lacking an S-nitrosyl or S-hydroxymethyl group that binds to the active site zinc atom, the affinity of inhibitors GSH and S-methylglutathione is reduced by 2-3 orders of magnitude compared to GSNO and HMGSH |
|
| 1.1.1.284 | more |
not inhibitory: N-ethylmaleimide |
|
| 1.1.1.284 | ATP |
- |
  |
