EC Number |
Natural Substrates |
---|
6.3.5.4 | ATP + L-Asp + L-Gln |
TaASN1 is dramatically induced by salinity, osmotic stress and exogenous abscisic acid |
6.3.5.4 | ATP + L-Asp + L-Gln |
TaASN2 transcripts are very low in all detected tissues and conditions and are only slightly induced by abscisic acid in roots |
6.3.5.4 | ATP + L-Asp + L-Gln |
the basic region leucine zipper protein ATF5, a transcriptional activator, stimulates asparagine promoter/reporter gene transcription via the nutrient-sensing response unit |
6.3.5.4 | ATP + L-Asp + L-Gln |
light, carbon and nitrogen availability control asparagine synthesis in sunflower by regulating three aspargine synthetase coding genes. HAS2 expression requires light and is positively affected by sucrose. HAS1 and HAS1.1 expression is dependent on nitrogen availability, while HAS2 transcripts are still found in N-starved plants. High ammonium level induces all three asparagine synthetase genes and partially reverts sucrose repression of HAS1 and HAS1.1 |
6.3.5.4 | ATP + L-aspartate + L-glutamine |
- |
6.3.5.4 | ATP + L-aspartate + L-glutamine |
glutamine is the in vivo nitrogen source |
6.3.5.4 | ATP + L-aspartate + L-glutamine |
the enzyme might play a functional role in nitrogen translocation from root to aerial organs in Phaseolus vulgaris |
6.3.5.4 | ATP + L-aspartate + L-glutamine + H2O |
- |
6.3.5.4 | ATP + L-aspartate + NH3 |
- |
6.3.5.4 | ATP + L-aspartate + NH3 |
asparagine biosynthesis proceeds by initial reaction of aspartate and ATP to yield a beta-aspartyl-AMP intermediate, in the presence of glutamine, ammonia released in the N-terminal active site reacts with beta-aspartyl-AMP to yield asparagine and AMP |