Protein Variants | Comment | Organism |
---|---|---|
additional information | construction of FAAH-/- mice | Mus musculus |
Organism | UniProt | Comment | Textmining |
---|---|---|---|
Homo sapiens | - |
- |
- |
Mus musculus | - |
- |
- |
Mus musculus C57/BL6J | - |
- |
- |
Source Tissue | Comment | Organism | Textmining |
---|---|---|---|
cardiomyocyte | primary | Homo sapiens | - |
myocardium | - |
Mus musculus | - |
myocardium | - |
Homo sapiens | - |
Synonyms | Comment | Organism |
---|---|---|
FAAH | - |
Mus musculus |
FAAH | - |
Homo sapiens |
General Information | Comment | Organism |
---|---|---|
malfunction | genetic deletion of FAAH is associated with enhanced acute doxorubicin-induced myocardial cell death and decreased survival, mechanism of the doxorubicin-induced myocardial cell death in FAAH+/+ and FAAH-/- mice, overview | Mus musculus |
metabolism | fatty acid amide hydrolase is a key regulator of endocannabinoid-induced myocardial tissue injury | Mus musculus |
metabolism | fatty acid amide hydrolase is a key regulator of endocannabinoid-induced myocardial tissue injury | Homo sapiens |
additional information | anandamide induces enhanced cell death in human cardiomyocytes pretreated by FAAH inhibitor, and enhances sensitivity to reactive oxygen species generation in inflammatory cells of FAAH knockouts | Homo sapiens |
additional information | anandamide induces enhanced cell death in human cardiomyocytes pretreated by FAAH inhibitor, and enhances sensitivity to reactive oxygen species generation in inflammatory cells of FAAH knockouts, it triggers concentration-dependent respiratory burst (ROS generation) in neutrophil granulocytes isolated from FAAH+/+ mice | Mus musculus |
physiological function | fatty acid amide hydrolase is a key regulator of endocannabinoid-induced myocardial tissue injury | Mus musculus |
physiological function | fatty acid amide hydrolase is a key regulator of endocannabinoid-induced myocardial tissue injury | Homo sapiens |