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Results 1 - 10 of 13 > >>
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EC Number Protein Variants Commentary Reference
Display the word mapDisplay the reaction diagram Show all sequences 2.4.1.150A169T naturally occuring mutation in the GCNT2 gene causing blood group I and cataract formation phenotype 759326
Display the word mapDisplay the reaction diagram Show all sequences 2.4.1.150G310D naturally occuring mutation in the GCNT2 gene causing blood group I and cataract formation phenotype 759326
Display the word mapDisplay the reaction diagram Show all sequences 2.4.1.150G334R naturally occuring mutation in the GCNT2 gene causing blood group I and cataract formation phenotype 759326
Display the word mapDisplay the reaction diagram Show all sequences 2.4.1.150G348E naturally occuring mutation in the GCNT2 gene causing blood group I and cataract formation phenotype 759326
Display the word mapDisplay the reaction diagram Show all sequences 2.4.1.150more CRISPR-mediated knockout of GCNT3 in pancreatic cancer cells 758993
Display the word mapDisplay the reaction diagram Show all sequences 2.4.1.150more CRISPR-mediated knockout of GCNT3 in pancreatic cancer cells. A significant increase in pancreas weight is observed in Kras mice compared with wild-type mice. Only 6% of the pancreata from Kras mice are free from PanIN lesions and PDAC, whereas 100% of pancreata are normal in wild-type mice -, 758993
Display the word mapDisplay the reaction diagram Show all sequences 2.4.1.150more GCNT3 downregulation by shGCNT3 7. GCNT3 overexpression reduces 5-fluorouracil resistance in colorectal cancer (CRC) cells. GCNT3 overexpression reduces proliferation, invasion and changes metabolic capacities of CRC cells. Genomic analysis of GCNT3 overexpression, overview 760171
Display the word mapDisplay the reaction diagram Show all sequences 2.4.1.150more knockdown of GCNT3 using GCNT3-specific small interfering RNAs: siGCNT3-1 (GCUACUGCGAGCUGUGUAUTT), and siGCNT3-2 (GCUCAGUGCCGUGGAAAUATT). Reduction of the expression of endogenous GCNT3 by siRNA transfection in SGC7901 and BGC823 cells. Epstein-Barr virus (EBV) miRNA miR-BART1-5p specifically targets GCNT3. miR-BART1-5p suppresses GCNT3 expression, cell proliferation, and migration in EBVnGC, and the MiR-BART1-5p inhibitor increases GCNT3 expression, cell proliferation, and migration in transfected GT39 and GT38 cells. NF-kappaB can activate the expression of multiple miR-BARTs, including miR-BART1-5p 760226
Display the word mapDisplay the reaction diagram Show all sequences 2.4.1.150more mutation experiments at the GT boxes of the promoters of IGnT A and B genes 638562
Display the word mapDisplay the reaction diagram Show all sequences 2.4.1.150N388R homozygous frameshift mutation c.1163_1166delATCA, p.(Asn388Argfs*20) is the cause of congenital cataract in two affected siblings, pleiotropic effect of the mutation causing congenital cataract and adult I blood group phenotype, overview 759326
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