EC Number |
Disease |
PubMed ID |
Title of Publication |
Category |
Confidence Level |
---|
3.6.4.13 | Acquired Immunodeficiency Syndrome |
14747468 |
On the roles of Saccharomyces cerevisiae Dna2p and Flap endonuclease 1 in Okazaki fragment processing. |
unassigned |
0 |
3.6.4.13 | Acquired Immunodeficiency Syndrome |
15910005 |
Escherichia coli DbpA is a 3' --> 5' RNA helicase. |
unassigned |
0 |
3.6.4.13 | Acquired Immunodeficiency Syndrome |
24630996 |
Helicase and polymerase move together close to the fork junction and copy DNA in one-nucleotide steps. |
causal interaction unassigned |
1 0 |
3.6.4.13 | Acquired Immunodeficiency Syndrome |
26544712 |
Connecting Replication and Repair: YoaA, a Helicase-Related Protein, Promotes Azidothymidine Tolerance through Association with Chi, an Accessory Clamp Loader Protein. |
therapeutic application unassigned |
1 0 |
3.6.4.13 | Acquired Immunodeficiency Syndrome |
28238534 |
Structural Insights into a Unique Dimeric DEAD-Box Helicase CshA that Promotes RNA Decay. |
causal interaction unassigned |
2 0 |
3.6.4.13 | Acquired Immunodeficiency Syndrome |
30966844 |
The roles of apoptosis, autophagy and unfolded protein response in arbovirus, influenza virus, and HIV infections. |
ongoing research unassigned |
2 0 |
3.6.4.13 | Acquired Immunodeficiency Syndrome |
31300642 |
The mechanism of RNA duplex recognition and unwinding by DEAD-box helicase DDX3X. |
causal interaction therapeutic application unassigned |
1 4 0 |
3.6.4.13 | Acquired Immunodeficiency Syndrome |
33966599 |
Lipotoxicity reduces DDX58/Rig-1 expression and activity leading to impaired autophagy and cell death. |
causal interaction ongoing research therapeutic application unassigned |
1 2 1 0 |
3.6.4.13 | Acro-Osteolysis |
34453469 |
A novel pathogenic variant p.Asp797Val in IFIH1 in a Japanese boy with overlapping Singleton-Merten syndrome and Aicardi-Goutières syndrome. |
causal interaction unassigned |
1 0 |
3.6.4.13 | Acute Lung Injury |
32504068 |
?-sitosterol ameliorates influenza A virus-induced proinflammatory response and acute lung injury in mice by disrupting the cross-talk between RIG-I and IFN/STAT signaling. |
causal interaction ongoing research therapeutic application unassigned |
3 2 4 0 |