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4.3.2.9
malfunction
enzyme knockdown inhibits glioma cell T98G and U251 proliferation and colony formation
735619
4.3.2.9
metabolism
isoform GGCT2;1 participates in the cellular response to arsenic via glutathione degradation. The enzyme ensures sufficient glutathione turnover during abiotic stress by recycling L-Glu. Enzyme overexpression provides enhanced arsenite tolerance in Arabidopsis and decreases nitrogen requirement under arsenite toxicity
736987
4.3.2.9
physiological function
enzyme overexpression promotes the expression of Notch receptors and activates Akt signaling in glioma cells
735619
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