EC Number   |
Expression |
Reference |
|---|
   2.4.2.31 | down |
mutating the DNA consensus sequence of either the E box or the A/T-rich element results in a nearly complete loss of ART1 promoter inducibility |
702912 |
| 2.4.2.31 | more |
Peptoclostridium difficile can be divided into at least five diffent toxin-producing groups: large clostridial cytotoxins producers, large clostridial cytotoxins and binary toxin producers, toxin B-only producers, toxin B and binary toxin producers and binary toxin-only producers. These groupings add further support to the theory Peptoclostridium difficile is divided into stable subpopulations that have evolved from common ancestors |
727540 |
| 2.4.2.31 | up |
Art7.1, but not Art7.2, is predominantly expressed on immune cells |
705674 |
| 2.4.2.31 | up |
enzyme expression is activated during the endoplasmic reticulum stress response |
735914 |
| 2.4.2.31 | up |
enzyme expression is increased in colon adenocarcinoma tissues |
736275 |
| 2.4.2.31 | up |
expression of ART1, ART3 and ART5 mRNA in myogenic cell lines during skeletal muscle differentiation in vitro. ART1 expression is restricted to myotube formation. ART5 similar to ART1 mRNA is strongly upregulated during muscle cell differentiation. Upregulation of ART3 mRNA only in C3H10T1/2 cells. Differentiation-dependent upregulation of ART1 mRNA is induced by the binding of myogenin to an E box and of MEF-2 to an A/T-rich element in the proximal promoter region of the ART1 gene |
702912 |
