EC Number   |
Expression |
Reference |
|---|
    2.4.1.133 | down |
both anti-TNFR1 and anti-TNFR2 antibodies suppressed beta-1,4-GalT I mRNA expression induced by tumor necrosis factor-alpha or lipopolysaccharide |
703134 |
| 2.4.1.133 | more |
decreasing of the expression of GalT I in hepatoma cells reduce the ability of tumor formation in vivo and inhibits hepatitis B virus-encoded protein HBx-induced cell cycle progression |
704930 |
| 2.4.1.133 | up |
beta-1,4-GalT I mRNA reaches peaks at 2 weeks after sciatic nerve crush, in situ hybridization shows that at day 1 after sciatic nerve crush, the expression levels of beta-1,4-GalT I mRNA is strong at the crush site, and decreases gradually from crush site to the distal segments, the expression of beta-1,4-GalTI mRNA is related to the injury site and injury time in peripheral nervous system |
705215 |
| 2.4.1.133 | up |
beta-1,4-GalT V mRNA reaches peaks at 2 weeks after sciatic nerve crush, in situ hybridization shows that at day 1 after sciatic nerve crush, the expression levels of beta-1,4-GalT V mRNA is strong at the crush site, and decreases gradually from crush site to the distal segments |
705215 |
| 2.4.1.133 | up |
beta-1,4-GalT-I is strongly induced in the ventral midbrain by intranigral injection of lipopolysaccharide, significant increase in beta-1,4-GalT-I mRNA at 8 h after focal injection of lipopolysaccharide, and lastes to 18 h, then decreases gradually |
705941 |
| 2.4.1.133 | up |
beta-1,4-GalT-I mRNA is significant increased as early as 2 h after lipopolysaccharide stimulation (1 microM/ml), upregulation in a time- and concentration-dependent manner |
704010 |
| 2.4.1.133 | up |
GalT I is highly expressed in hepatocellular carcinoma and transcriptionally up-regulated by hepatitis B virus-encoded HBx, and functions as a positive growth regulator in hepatoma cells |
704930 |
| 2.4.1.133 | up |
in injured spinal cords, enzyme is significantly increased after 8 h, beta-1,4-GalT-I mRNA reaches the peak at 1 day after spinal cord contusion, then gradually reduced from day 5 to day 7, recovers to the baseline level at 14 day |
704802 |
| 2.4.1.133 | up |
the beta-1,4-GalT I mRNA reaches peaks at 2 weeks after sciatic nerve crush and 3 days after sciatic nerve transection in a dose-dependent manner, in inflammation, lipopolysaccharide administration affects beta-1,4-GalT I mRNA expression in sciatic nerve in a time- and dose-dependent manner, and beta-1,4-GalT I mRNA is expressed mainly in Schwann cells |
704790 |
| 2.4.1.133 | up |
the expression level of beta-1,4-GalT I mRNA in cultured type-2 astrocytes is dose-dependent compared with the control group, it decreases at 0.1 ng/ml tumor necrosis factor-alpha, reaches at a peak at 1 ng/ml and then restores to normal level at 5 ng/ml. The expression in cultured type-2 astrocytes is time-dependent, it increases from 0.5 h, peaks at 4 h, then it decreases gradually to normal level after exposure to 1 ng/ml tumor necrosis factor-alpha. Lipopolysaccharide induces beta-1,4-GalT I mRNA expression in type-2 astrocytes in a time- and dose-dependent manner |
703134 |
