2.3.1.255	Prostatic Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27659526&form=6&db=m	Acetylation of androgen receptor by ARD1 promotes dissociation from HSP90 complex and prostate tumorigenesis.	causal interaction,ongoing research,unassigned	3,2,0	
2.3.1.255	Tuberculosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27353550&form=6&db=m	Biochemical evidence for relaxed substrate specificity of N?-acetyltransferase (Rv3420c/rimI) of Mycobacterium tuberculosis.	unassigned	-	
2.3.1.255	Tuberculosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31389995&form=6&db=m	Biophysical and functional characterizations of recombinant RimI acetyltransferase from Mycobacterium tuberculosis.	causal interaction,ongoing research,therapeutic application,unassigned	3,4,1,0	
2.3.1.255	Carcinoma, Hepatocellular	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23258102&form=6&db=m	Clinical implications of arrest-defective protein 1 expression in hepatocellular carcinoma: a novel predictor of microvascular invasion.	causal interaction,diagnostic usage,ongoing research,unassigned	4,3,2,0	
2.3.1.255	Colonic Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22261620&form=6&db=m	Combined Phenotype of 4 Markers Improves Prognostic Value of Patients With Colon Cancer.	diagnostic usage,ongoing research,unassigned	1,3,0	
2.3.1.255	Starvation	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25330189&form=6&db=m	daf-31 encodes the catalytic subunit of N alpha-acetyltransferase that regulates Caenorhabditis elegans development, metabolism and adult lifespan.	therapeutic application,unassigned	4,0	
2.3.1.255	Thyroid Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20178098&form=6&db=m	Depletion of the human N(alpha)-terminal acetyltransferase A (hNatA) induces p53-dependent apoptosis and p53-independent growth inhibition.	ongoing research,therapeutic application,unassigned	2,1,0	
2.3.1.255	Thyroid Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21351257&form=6&db=m	Depletion of the human N?-terminal acetyltransferase A induces p53-dependent apoptosis and p53-independent growth inhibition.	ongoing research,therapeutic application,unassigned	3,1,0	
2.3.1.255	Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23557624&form=6&db=m	Design, Synthesis, and Kinetic Characterization of Protein N-Terminal Acetyltransferase Inhibitors.	causal interaction,diagnostic usage,unassigned	4,1,0	
2.3.1.255	Breast Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10464641&form=6&db=m	Evaluation of genotype data in clinical risk assessment: methods and application to BRCA1, BRCA2, and N-acetyl transferase-2 genotypes in breast cancer.	causal interaction,diagnostic usage,therapeutic application,unassigned	3,3,3,0