2.3.1.255	Atherosclerosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27755984&form=6&db=m	miRNA-27b modulates endothelial cell angiogenesis by directly targeting Naa15 in atherogenesis.	unassigned	-	
2.3.1.255	Breast Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=10464641&form=6&db=m	Evaluation of genotype data in clinical risk assessment: methods and application to BRCA1, BRCA2, and N-acetyl transferase-2 genotypes in breast cancer.	causal interaction,diagnostic usage,therapeutic application,unassigned	3,3,3,0	
2.3.1.255	Carcinogenesis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18794801&form=6&db=m	Implication of human N-alpha-acetyltransferase 5 in cellular proliferation and carcinogenesis.	causal interaction,ongoing research,unassigned	2,2,0	
2.3.1.255	Carcinogenesis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20592467&form=6&db=m	hNaa10p contributes to tumorigenesis by facilitating DNMT1-mediated tumor suppressor gene silencing.	causal interaction,diagnostic usage,ongoing research,therapeutic application	1,1,2,1	
2.3.1.255	Carcinoma, Hepatocellular	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20419844&form=6&db=m	LOH analysis of genes around D4S2964 identifies ARD1B as a prognostic predictor of hepatocellular carcinoma.	causal interaction,diagnostic usage,therapeutic application,unassigned	3,4,2,0	
2.3.1.255	Carcinoma, Hepatocellular	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23258102&form=6&db=m	Clinical implications of arrest-defective protein 1 expression in hepatocellular carcinoma: a novel predictor of microvascular invasion.	causal interaction,diagnostic usage,ongoing research,unassigned	4,3,2,0	
2.3.1.255	Cardiomyopathy, Hypertrophic	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33103328&form=6&db=m	Variants in NAA15 cause pediatric hypertrophic cardiomyopathy.	causal interaction,diagnostic usage,unassigned	3,1,0	
2.3.1.255	Cleft Lip	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33103328&form=6&db=m	Variants in NAA15 cause pediatric hypertrophic cardiomyopathy.	causal interaction,diagnostic usage,unassigned	3,1,0	
2.3.1.255	Colonic Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22261620&form=6&db=m	Combined Phenotype of 4 Markers Improves Prognostic Value of Patients With Colon Cancer.	diagnostic usage,ongoing research,unassigned	1,3,0	
2.3.1.255	Heart Defects, Congenital	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29621621&form=6&db=m	Phenotypic consequences of gene disruption by a balanced de novo translocation involving SLC6A1 and NAA15.	causal interaction,unassigned	4,0