2.1.1.355	Adenocarcinoma	https://pubmed.ncbi.nlm.nih.gov/32360551/	SETDB1 Inhibits p53-Mediated Apoptosis and Is Required for Formation of Pancreatic Ductal Adenocarcinomas in Mice.	causal interaction	2	
2.1.1.355	Adenocarcinoma	https://pubmed.ncbi.nlm.nih.gov/32729861/	Histone methyltransferase G9a inhibitor-loaded redox-responsive nanoparticles for pancreatic ductal adenocarcinoma therapy.	causal interaction	4	
2.1.1.355	Adenocarcinoma	https://pubmed.ncbi.nlm.nih.gov/23395645/	Enhancer of Zeste Homolog 2 Silences MicroRNA-218 in Human Pancreatic Ductal Adenocarcinoma Cells by Inducing Formation of Heterochromatin.	causal interaction	4	
2.1.1.355	Adenocarcinoma	https://pubmed.ncbi.nlm.nih.gov/23807219/	Gossypol and an HMT G9a inhibitor act in synergy to induce cell death in pancreatic cancer cells.	causal interaction	4	
2.1.1.355	Adenocarcinoma	https://pubmed.ncbi.nlm.nih.gov/31398867/	Expression of the Major and Pro-Oncogenic H3K9 Lysine Methyltransferase SETDB1 in Non-Small Cell Lung Cancer.	causal interaction	1	
2.1.1.355	Adenocarcinoma	https://pubmed.ncbi.nlm.nih.gov/33639051/	Effects of miR-362 in Regulating the Proliferation, Invasion and Apoptosis of Gastric Cancer by Inhibiting the Expression of Tumor-Promoting Factor PRDM2.	causal interaction	4	
2.1.1.355	Adenocarcinoma	https://pubmed.ncbi.nlm.nih.gov/34094655/	SETDB1 in cancer: overexpression and its therapeutic implications.	causal interaction	4	
2.1.1.355	Adenocarcinoma	https://pubmed.ncbi.nlm.nih.gov/34413625/	Histone Methyltransferases Useful in Gastric Cancer Research.	causal interaction	3	
2.1.1.355	Adenocarcinoma	https://pubmed.ncbi.nlm.nih.gov/32360551/	SETDB1 Inhibits p53-Mediated Apoptosis and Is Required for Formation of Pancreatic Ductal Adenocarcinomas in Mice.	diagnostic usage	2	
2.1.1.355	Adenocarcinoma	https://pubmed.ncbi.nlm.nih.gov/23395645/	Enhancer of Zeste Homolog 2 Silences MicroRNA-218 in Human Pancreatic Ductal Adenocarcinoma Cells by Inducing Formation of Heterochromatin.	diagnostic usage	3