1.1.1.102	Carcinogenesis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15505035&form=6&db=m	Identification of target genes regulated by FOXC1 using nickel agarose-based chromatin enrichment.	causal interaction,therapeutic application,unassigned	2,1,0	
1.1.1.102	Ichthyosis	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34277909&form=6&db=m	Progressive symmetrical erythrokeratoderma manifesting as harlequin-like ichthyosis with severe thrombocytopenia secondary to a homozygous 3-ketodihydrosphingosine reductase mutation.	causal interaction,unassigned	2,0	
1.1.1.102	Leukemia	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34373586&form=6&db=m	3-Ketodihydrosphingosine reductase maintains ER homeostasis and unfolded protein response in leukemia.	diagnostic usage,ongoing research,therapeutic application,unassigned	1,2,1,0	
1.1.1.102	Lymphoma, Follicular	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8417785&form=6&db=m	FVT-1, a novel human transcription unit affected by variant translocation t(2;18)(p11;q21) of follicular lymphoma.	causal interaction,unassigned	3,0	
1.1.1.102	Muscular Atrophy, Spinal	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17420465&form=6&db=m	A missense mutation in the 3-ketodihydrosphingosine reductase FVT1 as candidate causal mutation for bovine spinal muscular atrophy.	causal interaction,unassigned	3,0	
1.1.1.102	Neoplasms	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8417785&form=6&db=m	FVT-1, a novel human transcription unit affected by variant translocation t(2;18)(p11;q21) of follicular lymphoma.	causal interaction,unassigned	3,0	
1.1.1.102	Thrombocytopenia	http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34277909&form=6&db=m	Progressive symmetrical erythrokeratoderma manifesting as harlequin-like ichthyosis with severe thrombocytopenia secondary to a homozygous 3-ketodihydrosphingosine reductase mutation.	causal interaction,unassigned	2,0