6.3.1.5 Asthma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=8568107&form=6&db=m Asthma and oxidant stress: nutritional, environmental, and genetic risk factors. causal interaction,therapeutic application,unassigned 4,1,0 6.3.1.5 Congenital Abnormalities http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28067697&form=6&db=m National Administrative Databases in Adult Spinal Deformity Surgery: A Cautionary Tale. causal interaction,therapeutic application,unassigned 1,2,0 6.3.1.5 Frontotemporal Dementia http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21965302&form=6&db=m NMNAT suppresses tau-induced neurodegeneration by promoting clearance of hyperphosphorylated tau oligomers in a Drosophila model of tauopathy. causal interaction,therapeutic application,unassigned 3,2,0 6.3.1.5 Glioma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=12777395&form=6&db=m The reported human NADsyn2 is ammonia-dependent NAD synthetase from a pseudomonad. ongoing research,unassigned 4,0 6.3.1.5 Hepatorenal Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27081787&form=6&db=m Nonabsorbable disaccharides for hepatic encephalopathy: A systematic review and meta-analysis. causal interaction,diagnostic usage,therapeutic application,unassigned 2,3,4,0 6.3.1.5 HIV Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33620297&form=6&db=m High population-attributable fractions of traditional risk factors for non-AIDS-defining diseases among people living with HIV in China: a cohort study. unassigned - 6.3.1.5 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24191058&form=6&db=m A genetic strategy to identify targets for the development of drugs that prevent bacterial persistence. ongoing research,unassigned 4,0 6.3.1.5 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27081787&form=6&db=m Nonabsorbable disaccharides for hepatic encephalopathy: A systematic review and meta-analysis. causal interaction,diagnostic usage,therapeutic application,unassigned 2,3,4,0 6.3.1.5 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32743503&form=6&db=m Identifying the culprits in neurological autoimmune diseases. causal interaction,diagnostic usage,unassigned 3,2,0 6.3.1.5 Liver Failure http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27081787&form=6&db=m Nonabsorbable disaccharides for hepatic encephalopathy: A systematic review and meta-analysis. causal interaction,diagnostic usage,therapeutic application,unassigned 2,3,4,0 6.3.1.5 Multiple Sclerosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32743503&form=6&db=m Identifying the culprits in neurological autoimmune diseases. causal interaction,diagnostic usage,unassigned 3,2,0 6.3.1.5 Myasthenia Gravis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32743503&form=6&db=m Identifying the culprits in neurological autoimmune diseases. causal interaction,diagnostic usage,unassigned 3,2,0 6.3.1.5 Neuroaxonal Dystrophies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23622415&form=6&db=m Axonal dystrophies. therapeutic application,unassigned 1,0 6.3.1.5 Neuromyelitis Optica http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32743503&form=6&db=m Identifying the culprits in neurological autoimmune diseases. causal interaction,diagnostic usage,unassigned 3,2,0 6.3.1.5 Ovarian Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34314702&form=6&db=m Ribosome ADP-ribosylation inhibits translation and maintains proteostasis in cancers. causal interaction,unassigned 4,0 6.3.1.5 Parkinsonian Disorders http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21965302&form=6&db=m NMNAT suppresses tau-induced neurodegeneration by promoting clearance of hyperphosphorylated tau oligomers in a Drosophila model of tauopathy. causal interaction,therapeutic application,unassigned 3,2,0 6.3.1.5 Peritonitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27081787&form=6&db=m Nonabsorbable disaccharides for hepatic encephalopathy: A systematic review and meta-analysis. causal interaction,diagnostic usage,therapeutic application,unassigned 2,3,4,0 6.3.1.5 Starvation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18490451&form=6&db=m Biosynthesis and recycling of nicotinamide cofactors in mycobacterium tuberculosis. An essential role for NAD in nonreplicating bacilli. unassigned - 6.3.1.5 Tauopathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32250733&form=6&db=m Nicotinamide mononucleotide adenylyltransferase uses its NAD+ substrate-binding site to chaperone phosphorylated Tau. causal interaction,unassigned 1,0 6.3.1.5 Tuberculosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=9620974&form=6&db=m The MTCY428.08 gene of Mycobacterium tuberculosis codes for NAD+ synthetase. causal interaction,ongoing research,therapeutic application,unassigned 2,1,1,0 6.3.1.5 Tuberculosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=15748981&form=6&db=m Glutamine amidotransferase activity of NAD+ synthetase from Mycobacterium tuberculosis depends on an amino-terminal nitrilase domain. ongoing research,unassigned 3,0 6.3.1.5 Tuberculosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24191058&form=6&db=m A genetic strategy to identify targets for the development of drugs that prevent bacterial persistence. ongoing research,unassigned 4,0 6.3.1.5 Tuberculosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31911602&form=6&db=m Different ways to transport ammonia in human and Mycobacterium tuberculosis NAD+ synthetases. ongoing research,therapeutic application,unassigned 2,1,0