1.13.11.18 Acquired Immunodeficiency Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31120291&form=6&db=m Intergroup contact, social dominance, and environmental concern: A test of the cognitive-liberalization hypothesis. causal interaction,unassigned 2,0 1.13.11.18 Adenomatous Polyposis Coli http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31258845&form=6&db=m ETHE1 overexpression promotes SIRT1 and PGC1? mediated aerobic glycolysis, oxidative phosphorylation, mitochondrial biogenesis and colorectal cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,1,4,4 1.13.11.18 Anemia, Hypochromic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24692429&form=6&db=m The mitochondrial sulfur dioxygenase ETHE1 is required for amino acid catabolism during carbohydrate starvation and embryo development in Arabidopsis thaliana. causal interaction,unassigned 1,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=14732903&form=6&db=m Ethylmalonic encephalopathy is caused by mutations in ETHE1, a gene encoding a mitochondrial matrix protein. causal interaction,unassigned 3,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16183799&form=6&db=m ETHE1 mutations are specific to ethylmalonic encephalopathy. causal interaction,therapeutic application,unassigned 4,1,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=16929096&form=6&db=m Structure of an ETHE1-like protein from Arabidopsis thaliana. causal interaction,unassigned 4,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18593870&form=6&db=m Identification of new mutations in the ETHE1 gene in a cohort of 14 patients presenting with ethylmalonic encephalopathy. causal interaction,unassigned 2,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=18656261&form=6&db=m Spectroscopic studies on Arabidopsis ETHE1, a glyoxalase II-like protein. causal interaction,therapeutic application,unassigned 3,1,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19136963&form=6&db=m Loss of ETHE1, a mitochondrial dioxygenase, causes fatal sulfide toxicity in ethylmalonic encephalopathy. causal interaction,therapeutic application,unassigned 2,1,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20657580&form=6&db=m Combined treatment with oral metronidazole and N-acetylcysteine is effective in ethylmalonic encephalopathy. causal interaction,unassigned 4,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20812865&form=6&db=m Chronic Exposure to Sulfide causes Accelerated Degradation of Cytochrome c oxidase in Ethylmalonic Encephalopathy. causal interaction,ongoing research,therapeutic application,unassigned 4,4,1,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20978941&form=6&db=m Multiple sources of metabolic disturbance in ETHE1-related ethylmalonic encephalopathy. causal interaction,unassigned 4,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21410200&form=6&db=m Proteomics Reveals that Redox Regulation Is Disrupted in Patients with Ethylmalonic Encephalopathy. causal interaction,unassigned 4,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=21472225&form=6&db=m Novel ETHE1 mutation in a carrier couple having prior offspring affected with ethylmalonic encephalopathy: Genetic analysis, clinical management and reproductive outcome. causal interaction,unassigned 3,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22584649&form=6&db=m Ethylmalonic encephalopathy associated with crescentic glomerulonephritis. causal interaction,unassigned 4,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22786886&form=6&db=m Arabidopsis ETHE1 Encodes a Sulfur Dioxygenase That Is Essential for Embryo and Endosperm Development. unassigned - 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=23144459&form=6&db=m Characterization of patient mutations in human persulfide dioxygenase (ETHE1) involved in H2S catabolism. causal interaction,unassigned 2,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24389926&form=6&db=m Distribution, diversity, and activities of sulfur dioxygenases in heterotrophic bacteria. causal interaction,ongoing research,unassigned 1,2,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24410953&form=6&db=m Expression of abiotic stress inducible ETHE1-like protein from rice is higher in roots and is regulated by calcium. causal interaction,unassigned 2,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25198162&form=6&db=m Ethylmalonic encephalopathy ETHE1 R163W/R163Q mutations alter protein stability and redox properties of the iron centre. causal interaction,unassigned 4,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25482779&form=6&db=m Stress response of OsETHE1 is altered in response to light and dark conditions. causal interaction,unassigned 3,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25596185&form=6&db=m Crystal structure of human persulfide dioxygenase: structural basis of ethylmalonic encephalopathy. unassigned - 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25747485&form=6&db=m Sulfide detoxification in plant mitochondria. causal interaction,unassigned 1,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26177047&form=6&db=m Staphylococcus aureus CstB Is a Novel Multidomain Persulfide Dioxygenase-Sulfurtransferase Involved in Hydrogen Sulfide Detoxification. therapeutic application,unassigned 1,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26194623&form=6&db=m Severe early onset ethylmalonic encephalopathy with West syndrome. causal interaction,therapeutic application,unassigned 4,1,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26194912&form=6&db=m Mitochondrial diseases caused by toxic compound accumulation: from etiopathology to therapeutic approaches. unassigned - 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26867521&form=6&db=m Quantitative proteomics suggests metabolic reprogramming during ETHE1 deficiency. causal interaction,unassigned 4,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27074420&form=6&db=m Untargeted Metabolomics Analysis Reveals a Link between ETHE1-Mediated Disruptive Redox State and Altered Metabolic Regulation. causal interaction,therapeutic application,unassigned 4,4,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27742479&form=6&db=m Protein polysulfidation-dependent persulfide dioxygenase activity of ethylmalonic encephalopathy protein 1. unassigned - 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29526615&form=6&db=m Response to medical and a novel dietary treatment in newborn screen identified patients with ethylmalonic encephalopathy. causal interaction,unassigned 2,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30298498&form=6&db=m [Clinical and genetic analysis of a case with atypical ethyl malonate encephalopathy]. causal interaction,diagnostic usage,unassigned 2,2,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30349987&form=6&db=m Acute and Chronic Management in an Atypical Case of Ethylmalonic Encephalopathy. causal interaction,unassigned 4,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30391543&form=6&db=m Deficiency of the mitochondrial sulfide regulator ETHE1 disturbs cell growth, glutathione level and causes proteome alterations outside mitochondria. causal interaction,unassigned 4,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30864297&form=6&db=m Improved clinical outcome following liver transplant in patients with ethylmalonic encephalopathy. causal interaction,unassigned 3,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31258845&form=6&db=m ETHE1 overexpression promotes SIRT1 and PGC1? mediated aerobic glycolysis, oxidative phosphorylation, mitochondrial biogenesis and colorectal cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,1,4,4 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32362910&form=6&db=m Novel Compound Heterozygous Variants of ETHE1 Causing Ethylmalonic Encephalopathy in a Chinese Patient: A Case Report. causal interaction,unassigned 3,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32485156&form=6&db=m Identification of a novel homozygous nonsense variant in a Chinese patient with ethylmalonic encephalopathy and a genotype-phenotype spectrum review. causal interaction,unassigned 3,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32923369&form=6&db=m Ethylmalonic encephalopathy: Clinical course and therapy response in an uncommon mild case with a severe ETHE1 mutation. causal interaction,therapeutic application,unassigned 4,4,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33034777&form=6&db=m Mitochondrial Dysfunction and Redox Homeostasis Impairment as Pathomechanisms of Brain Damage in Ethylmalonic Encephalopathy: Insights from Animal and Human Studies. causal interaction,unassigned 4,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33177801&form=6&db=m Compromised therapeutic value of pediatric liver transplantation in ethylmalonic encephalopathy: A case report. causal interaction,unassigned 4,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33639274&form=6&db=m Ethylmalonic encephalopathy ETHE1 p. D165H mutation alters the mitochondrial function in human skeletal muscle proteome. causal interaction,ongoing research,unassigned 1,2,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33900143&form=6&db=m Acute Strokelike Presentation and Long-term Evolution of Diffusion Restriction Pattern in Ethylmalonic Encephalopathy. causal interaction,unassigned 3,0 1.13.11.18 Brain Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=100013029&form=6&db=m Expression of abiotic stress inducible ETHE1?like protein from rice is higher in roots and is regulated by calcium causal interaction,unassigned 2,0 1.13.11.18 Carcinogenesis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31258845&form=6&db=m ETHE1 overexpression promotes SIRT1 and PGC1? mediated aerobic glycolysis, oxidative phosphorylation, mitochondrial biogenesis and colorectal cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,1,4,4 1.13.11.18 Colorectal Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31258845&form=6&db=m ETHE1 overexpression promotes SIRT1 and PGC1? mediated aerobic glycolysis, oxidative phosphorylation, mitochondrial biogenesis and colorectal cancer. causal interaction,diagnostic usage,ongoing research,therapeutic application 4,1,4,4 1.13.11.18 Colorectal Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34249670&form=6&db=m Systems Biomedicine of Primary and Metastatic Colorectal Cancer Reveals Potential Therapeutic Targets. causal interaction,diagnostic usage,unassigned 2,1,0 1.13.11.18 Congenital Abnormalities http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=17823034&form=6&db=m Total hip arthroplasty requiring subtrochanteric osteotomy for developmental hip dysplasia 5- to 14-year results. unassigned - 1.13.11.18 COVID-19 http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33745729&form=6&db=m U.S. public support for COVID-19 vaccine donation to low- and middle-income countries during the COVID-19 pandemic. causal interaction,diagnostic usage,therapeutic application,unassigned 1,1,1,0 1.13.11.18 Craniocerebral Trauma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31547202&form=6&db=m Increased Risks of Suicide Attempt and Suicidal Drug Overdose Following Admission for Head Injury in Patients with Depression. causal interaction,diagnostic usage,unassigned 2,1,0 1.13.11.18 Craniocerebral Trauma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33362590&form=6&db=m Increasing Risks of Suicide Attempt and Suicidal Drug Overdose After Head Trauma in Patients With Sleep-Disordered Breathing: A Population-Based Study. causal interaction,therapeutic application,unassigned 2,1,0 1.13.11.18 Craniosynostoses http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26495188&form=6&db=m Piezosurgical Suturectomy and Sutural Distraction Osteogenesis for the Treatment of Unilateral Coronal Synostosis. causal interaction,ongoing research,therapeutic application,unassigned 4,1,1,0 1.13.11.18 Cytochrome-c Oxidase Deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20812865&form=6&db=m Chronic Exposure to Sulfide causes Accelerated Degradation of Cytochrome c oxidase in Ethylmalonic Encephalopathy. causal interaction,causal interaction,ongoing research,ongoing research,therapeutic application,therapeutic application,unassigned,unassigned 4,4,4,4,1,1,0,0 1.13.11.18 Diabetic Nephropathies http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27582557&form=6&db=m Evaluating Pharmacological Effects of Two Major Components of Shuangdan Oral Liquid: Role of Danshensu and Paeonol in Diabetic Nephropathy Rat. unassigned - 1.13.11.18 Enteritis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19385333&form=6&db=m [Epidemiological impact of RV gastroenteritis in the Abruzzo Region: SDO analysis] ongoing research,unassigned 1,0 1.13.11.18 Epilepsy http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31731830&form=6&db=m Differences in Incidence and Risks of Suicide Attempt and Suicidal Drug Overdose between Patients with Epilepsy with and without Comorbid Depression. causal interaction,diagnostic usage,unassigned 4,3,0 1.13.11.18 Gastroenteritis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=19385333&form=6&db=m [Epidemiological impact of RV gastroenteritis in the Abruzzo Region: SDO analysis] ongoing research,unassigned 1,0 1.13.11.18 Hyperlipidemias http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30882913&form=6&db=m Metabolomics analysis of the therapeutic mechanism of Semen Descurainiae Oil on hyperlipidemia rats using 1 H-NMR and LC-MS. causal interaction,therapeutic application,unassigned 1,1,0 1.13.11.18 Hypospadias http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32653308&form=6&db=m Hypospadias prevalence in the Emilia Romagna Region registry: Increasing or methodology? therapeutic application,unassigned 1,0 1.13.11.18 Infections http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24382268&form=6&db=m The role of short-chain dehydrogenase/oxidoreductase, induced by salt stress, on host interaction of B. pseudomallei. unassigned - 1.13.11.18 Melanoma http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33614670&form=6&db=m Second Diagnostic Opinion by Experienced Dermatopathologists in the Setting of a Referral Regional Melanoma Unit Significantly Improves the Clinical Management of Patients With Cutaneous Melanoma. causal interaction,therapeutic application,unassigned 1,3,0 1.13.11.18 Melioidosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32219018&form=6&db=m Altered proteome of a Burkholderia pseudomallei mutant defective in short-chain dehydrogenase affects cell adhesion, biofilm formation and heat stress tolerance. causal interaction,unassigned 4,0 1.13.11.18 Metabolic Syndrome http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=20332082&form=6&db=m Lipogenesis is decreased by grape seed proanthocyanidins according to liver proteomics of rats fed a high fat diet. causal interaction,therapeutic application,unassigned 3,4,0 1.13.11.18 Mitochondrial Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26194912&form=6&db=m Mitochondrial diseases caused by toxic compound accumulation: from etiopathology to therapeutic approaches. unassigned - 1.13.11.18 Mitochondrial Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30864297&form=6&db=m Improved clinical outcome following liver transplant in patients with ethylmalonic encephalopathy. causal interaction,unassigned 3,0 1.13.11.18 Mitochondrial Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33900143&form=6&db=m Acute Strokelike Presentation and Long-term Evolution of Diffusion Restriction Pattern in Ethylmalonic Encephalopathy. causal interaction,unassigned 3,0 1.13.11.18 Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28929714&form=6&db=m [Extending traceability of malignant testicular tumours using hospital discharge records: an experience in Veneto Region (Northern Italy)]. diagnostic usage,unassigned 2,0 1.13.11.18 Neurodegenerative Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29526615&form=6&db=m Response to medical and a novel dietary treatment in newborn screen identified patients with ethylmalonic encephalopathy. causal interaction,unassigned 2,0 1.13.11.18 Neurodegenerative Diseases http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32485156&form=6&db=m Identification of a novel homozygous nonsense variant in a Chinese patient with ethylmalonic encephalopathy and a genotype-phenotype spectrum review. causal interaction,unassigned 3,0 1.13.11.18 Obesity http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22531085&form=6&db=m Obesity discrimination: the role of physical appearance, personal ideology, and anti-fat prejudice. therapeutic application,unassigned 1,0 1.13.11.18 Ovarian Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=33038519&form=6&db=m Salpingectomy for the Risk Reduction of Ovarian Cancer: Is It Time for a Salpingectomy-alone Approach? causal interaction,therapeutic application,unassigned 3,4,0 1.13.11.18 Overweight http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34525213&form=6&db=m Feminist identification, social dominance orientation, and weight bias in men. causal interaction,unassigned 3,0 1.13.11.18 Paraparesis, Spastic http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32923369&form=6&db=m Ethylmalonic encephalopathy: Clinical course and therapy response in an uncommon mild case with a severe ETHE1 mutation. causal interaction,therapeutic application,unassigned 4,4,0 1.13.11.18 Parkinson Disease http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=34308287&form=6&db=m Spatiotemporal characteristics of postsaccadic dynamic overshoot in young and elderly subjects. causal interaction,therapeutic application,unassigned 3,1,0 1.13.11.18 persulfide dioxygenase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22020834&form=6&db=m Morphologic evidence of diffuse vascular damage in human and in the experimental model of ethylmalonic encephalopathy. causal interaction,therapeutic application,unassigned 4,1,0 1.13.11.18 persulfide dioxygenase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26867521&form=6&db=m Quantitative proteomics suggests metabolic reprogramming during ETHE1 deficiency. causal interaction,unassigned 4,0 1.13.11.18 persulfide dioxygenase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=27074420&form=6&db=m Untargeted Metabolomics Analysis Reveals a Link between ETHE1-Mediated Disruptive Redox State and Altered Metabolic Regulation. causal interaction,therapeutic application,unassigned 4,4,0 1.13.11.18 persulfide dioxygenase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30056533&form=6&db=m Evidence that Thiosulfate Inhibits Creatine Kinase Activity in Rat Striatum via Thiol Group Oxidation. therapeutic application,unassigned 1,0 1.13.11.18 persulfide dioxygenase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30283487&form=6&db=m The Role of Persulfide Metabolism During Arabidopsis Seed Development Under Light and Dark Conditions. causal interaction,unassigned 3,0 1.13.11.18 persulfide dioxygenase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30391543&form=6&db=m Deficiency of the mitochondrial sulfide regulator ETHE1 disturbs cell growth, glutathione level and causes proteome alterations outside mitochondria. causal interaction,unassigned 4,0 1.13.11.18 persulfide dioxygenase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30399433&form=6&db=m Evidence that thiol group modification and reactive oxygen species are involved in hydrogen sulfide-induced mitochondrial permeability transition pore opening in rat cerebellum. causal interaction,therapeutic application,unassigned 2,1,0 1.13.11.18 persulfide dioxygenase deficiency http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=31477743&form=6&db=m ETHE1 and MOCS1 deficiencies: Disruption of mitochondrial bioenergetics, dynamics, redox homeostasis and endoplasmic reticulum-mitochondria crosstalk in patient fibroblasts. causal interaction,unassigned 4,0 1.13.11.18 Pulmonary Disease, Chronic Obstructive http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=32351414&form=6&db=m Suicide Attempt and Suicidal Drug Overdose in Chronic Obstructive Pulmonary Disease Patients With or Without Depression. causal interaction,diagnostic usage,therapeutic application,unassigned 4,3,3,0 1.13.11.18 Purpura http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=22584649&form=6&db=m Ethylmalonic encephalopathy associated with crescentic glomerulonephritis. causal interaction,unassigned 4,0 1.13.11.18 Starvation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=24692429&form=6&db=m The mitochondrial sulfur dioxygenase ETHE1 is required for amino acid catabolism during carbohydrate starvation and embryo development in Arabidopsis thaliana. causal interaction,unassigned 1,0 1.13.11.18 Starvation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=25482779&form=6&db=m Stress response of OsETHE1 is altered in response to light and dark conditions. causal interaction,unassigned 3,0 1.13.11.18 Starvation http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=30283487&form=6&db=m The Role of Persulfide Metabolism During Arabidopsis Seed Development Under Light and Dark Conditions. causal interaction,unassigned 3,0 1.13.11.18 Stroke http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29445281&form=6&db=m Suicidal drug overdose following stroke in elderly patients: a retrospective population-based cohort study. causal interaction,diagnostic usage,ongoing research,unassigned 3,4,1,0 1.13.11.18 Synostosis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=26495188&form=6&db=m Piezosurgical Suturectomy and Sutural Distraction Osteogenesis for the Treatment of Unilateral Coronal Synostosis. causal interaction,ongoing research,therapeutic application,unassigned 4,1,1,0 1.13.11.18 Synovitis http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=29968759&form=6&db=m Identification of pathways and genes associated with synovitis in osteoarthritis using bioinformatics analyses. unassigned - 1.13.11.18 Testicular Neoplasms http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?uid=28929714&form=6&db=m [Extending traceability of malignant testicular tumours using hospital discharge records: an experience in Veneto Region (Northern Italy)]. diagnostic usage,unassigned 2,0