5.5.1.24	malfunction	a mutant deficient in tocopherol cyclase is totally devoid of tocopherol. Tocopherol deficiency results in the increase in ascorbate and glutathione. The simultaneous loss of tocopherol and glutathione in a tocopherol cyclase/cad2 double deletion mutant results in moderate oxidative stress that affects the stability and the efficiency of the photosynthetic apparatus (reduced growth, chlorophyll content, and photosynthetic quantum)	728508	
5.5.1.24	malfunction	deletion of the enzyme leads to tocopherol deficiency and 2,3-dimethyl-6-phytylbenzene-1,4-diol accumulation	728506	
5.5.1.24	malfunction	the inactivation of vte1 genes results in increased guaiacol peroxidase activity and 1.4fold higher concentrations of total and oxidized glutathione and 1.2-1.3fold higher [GSSG]/[total GSH] ratio. Ascorbate peroxidase activity is lower in the tocopherol-deficient mutant vte1 than that in wild type plants	728496	
5.5.1.24	malfunction	transgenic plants with VTE1 RNA interference are more sensitive to salt stress (treatment with 100 mM NaCl for 10 days)	728794	
5.5.1.24	metabolism	key enzyme of tocopherol (vitamin E) synthesis	727854	
5.5.1.24	metabolism	the enzyme catalyzes the penultimate step of tocopherol synthesis	727218	
5.5.1.24	metabolism	the enzyme is involved in prenylquinone metabolism	747864	
5.5.1.24	metabolism	tocopherol cyclase catalyzes the penultimate step of tocopherol synthesis	728508	
5.5.1.24	physiological function	enzyme overexpression can increase the tolerance to drought stress. The increase in tocopherol affects cellular signaling and antioxidant defense of plants in response to drought	748909	
5.5.1.24	physiological function	overexpression of the enzyme increases the antioxidant capacity of rice seedlings. Overexpression of the enzyme is able to efficiently eliminate H2O2 produced by salt stress	728794