Literature summary extracted from

Dunger, G.; Relling, V.M.; Tondo, M.L.; Barreras, M.; Ielpi, L.; Orellano, E.G.; Ottado, J.
Xanthan is not essential for pathogenicity in citrus canker but contributes to Xanthomonas epiphytic survival (2007), Arch. Microbiol., 188, 127-135.

Organism

EC Number	Organism	UniProt	Comment	Textmining
2.7.8.31	Xanthomonas axonopodis	-	pv. citri	-

General Information

EC Number	General Information	Comment	Organism
2.7.8.31	malfunction	xanthan-deficient mutants of Xanthomonas axonopodis pv. citri, the bacterium responsible for citrus canker, are generated by deletion and marker exchange of the region encoding the carboxy-terminal end of the first glycosyltransferase, GumD. Mutants of gumD do not produce xanthan and remain pathogenic in citrus plants to the same extent as wild-type bacteria. The kinetics of appearance of initial symptoms, areas of plant material affected, and growth of bacteria inside plant tissue throughout the disease process are similar for both wild-type and mutant inoculations. Exopolysaccharide deffciency does not impair the ability of the bacteria to induce hypersensitive response on non-host plants. Apart from variations in phenotypic aspects, no differences in growth or survival under diVerent stress conditions are observed between the xanthan-deficient mutant and wild-type bacteria. gumD mutants display impaired survival under oxidative stress during stationary phase as well as impaired epiphytic survival on citrus leaves	Xanthomonas axonopodis

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Release 2023.1 (January 2023)