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Literature summary for 7.2.2.9 extracted from

  • Quian, Y.; Tiffany-Castiglioni, E.; Welsh, J.; Harris, E.D.
    Copper efflux from murine microvascular cells requires expression of the Menkes disease Cu-ATPase (1998), J. Nutr., 128, 1276-1282.
    View publication on PubMed

Natural Substrates/ Products (Substrates)

Natural Substrates Organism Comment (Nat. Sub.) Natural Products Comment (Nat. Pro.) Rev. Reac.
ATP + H2O + Cu2+/in Mus musculus
-
ADP + phosphate + Cu2+/out
-
?
ATP + H2O + Cu2+/in Homo sapiens Cu-ATPase in the blood-brain barrier controls the penetration of Cu into the brain, lesions to the Cu-ATPase in cerebrovascular endothelial cells are a primary cause of low brain Cu levels in Menkes disease ADP + phosphate + Cu2+/out
-
?

Organism

Organism UniProt Comment Textmining
Homo sapiens
-
-
-
Mus musculus
-
-
-

Source Tissue

Source Tissue Comment Organism Textmining
cerebrovascular endothelial cell
-
Mus musculus
-

Substrates and Products (Substrate)

Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
ATP + H2O + Cu2+/in
-
Mus musculus ADP + phosphate + Cu2+/out
-
?
ATP + H2O + Cu2+/in
-
Homo sapiens ADP + phosphate + Cu2+/out
-
?
ATP + H2O + Cu2+/in Cu-ATPase in the blood-brain barrier controls the penetration of Cu into the brain, lesions to the Cu-ATPase in cerebrovascular endothelial cells are a primary cause of low brain Cu levels in Menkes disease Homo sapiens ADP + phosphate + Cu2+/out
-
?