Data extracted from this reference:
Cloned(Commentary)
gene cop4
Arabidopsis thaliana
Protein Variants
additional information
generation of a cop4 defective mutant and a cop4/cop1 double mutant
Arabidopsis thaliana
Natural Substrates/ Products (Substrates)
(2E,6E)-farnesyl diphosphate
Arabidopsis thaliana
(+)-sativene + diphosphate
?
Organism
Arabidopsis thaliana
gene cop4
Source Tissue
seedling
Arabidopsis thaliana
Substrates and Products (Substrate)
(2E,6E)-farnesyl diphosphate
720695
Arabidopsis thaliana
(+)-sativene + diphosphate
?
Synonyms
Cop4
Arabidopsis thaliana
Cloned(Commentary) (protein specific)
gene cop4
Arabidopsis thaliana
Engineering (protein specific)
additional information
generation of a cop4 defective mutant and a cop4/cop1 double mutant
Arabidopsis thaliana
Natural Substrates/ Products (Substrates) (protein specific)
(2E,6E)-farnesyl diphosphate
Arabidopsis thaliana
(+)-sativene + diphosphate
?
Source Tissue (protein specific)
seedling
Arabidopsis thaliana
Substrates and Products (Substrate) (protein specific)
(2E,6E)-farnesyl diphosphate
720695
Arabidopsis thaliana
(+)-sativene + diphosphate
?
General Information
malfunction
identification of constitutive photomorphogenic mutants, cop2, cop3, and cop4,in which dark-grown seedlings have open and enlarged cotyledons resembling those of light-grown wild-type seedlings. Mutations in each of the three loci alleviate the normal inhibition of cell-type differentiation, cell enlargement, and lateral cell division observed in cotyledons of dark-grown wild-type seedlings, but do not affect plastid differentiation. Cop4 mutation also leads to high-leve1 dark expression of nuclear, but not plastid-encoded, light-inducible genes
Arabidopsis thaliana
metabolism
epistatic relationships of these three mutations to previously characterized phytochrome-deficient mutations suggest that COP2, COP3, and COP4 may act downstream of phytochrome in the light regulatory pathway
Arabidopsis thaliana
physiological function
the COP4 locus may be involved in both light-signaling and gravity-sensing processes. COP4 modulates cabl promoter activity through a pathway distinct from that of COPl and COP9, nuclear cabl gene encodes a chlorophyll alb binding protein of the photosynthetic light-harvesting complex. Modulation of cabl promoter activity by light and by the cop4 mutation
Arabidopsis thaliana
General Information (protein specific)
malfunction
identification of constitutive photomorphogenic mutants, cop2, cop3, and cop4,in which dark-grown seedlings have open and enlarged cotyledons resembling those of light-grown wild-type seedlings. Mutations in each of the three loci alleviate the normal inhibition of cell-type differentiation, cell enlargement, and lateral cell division observed in cotyledons of dark-grown wild-type seedlings, but do not affect plastid differentiation. Cop4 mutation also leads to high-leve1 dark expression of nuclear, but not plastid-encoded, light-inducible genes
Arabidopsis thaliana
metabolism
epistatic relationships of these three mutations to previously characterized phytochrome-deficient mutations suggest that COP2, COP3, and COP4 may act downstream of phytochrome in the light regulatory pathway
Arabidopsis thaliana
physiological function
the COP4 locus may be involved in both light-signaling and gravity-sensing processes. COP4 modulates cabl promoter activity through a pathway distinct from that of COPl and COP9, nuclear cabl gene encodes a chlorophyll alb binding protein of the photosynthetic light-harvesting complex. Modulation of cabl promoter activity by light and by the cop4 mutation
Arabidopsis thaliana
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