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Literature summary for 3.5.3.18 extracted from

  • Chen, Y.; Li, Y.; Zhang, P.; Traverse, J.H.; Hou, M.; Xu, X.; Kimoto, M.; Bache, R.J.
    Dimethylarginine dimethylaminohydrolase and endothelial dysfunction in failing hearts (2005), Am. J. Physiol. Heart Circ. Physiol., 289, H2212-2219.
    View publication on PubMed

Application

Application Comment Organism
medicine decreased DDAH-1 expression may cause accumulation of endogenous inhibitors of enothelial NO synthase, thereby contributing to endothelial dysfunction in the failing heart Homo sapiens
medicine decreased DDAH-1 expression may cause accumulation of endogenous inhibitors of enothelial NO synthase, thereby contributing to endothelial dysfunction in the failing heart Canis lupus familiaris

Localization

Localization Comment Organism GeneOntology No. Textmining
cytosol
-
Homo sapiens 5829
-
cytosol cardiac myocytes Canis lupus familiaris 5829
-
nucleus
-
Homo sapiens 5634
-
sarcolemma cardiac myocytes Canis lupus familiaris 42383
-

Organism

Organism UniProt Comment Textmining
Canis lupus familiaris
-
adult mongrels
-
Homo sapiens
-
-
-

Source Tissue

Source Tissue Comment Organism Textmining
cardiac myocyte weak staining Canis lupus familiaris
-
endothelial cell human microvascular and ubilical vein endothelial cells Homo sapiens
-
endothelium coronary microvessels, arteries and veins, strong staining Canis lupus familiaris
-
myocardium
-
Canis lupus familiaris
-
smooth muscle strong staining Canis lupus familiaris
-

Synonyms

Synonyms Comment Organism
DDAH-1
-
Homo sapiens
DDAH-1
-
Canis lupus familiaris
dimethylarginine dimethylaminohydrolase
-
Homo sapiens
dimethylarginine dimethylaminohydrolase
-
Canis lupus familiaris