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Literature summary for 3.4.21.9 extracted from

  • Mann, N.S.; Mann, S.K.
    Enterokinase (1994), Proc. Soc. Exp. Biol. Med., 206, 114-118.
    View publication on PubMed

Natural Substrates/ Products (Substrates)

Natural Substrates Organism Comment (Nat. Sub.) Natural Products Comment (Nat. Pro.) Rev. Reac.
Trypsinogen + H2O Homo sapiens enterokinase deficiency is a distinct clinical entity characterized by diarrhea, failure to thrive, hypoproteinemia, and edema. Acquired enterokinase deficiency may occur in some diffuse small bowel diseases. Steatorhea of cellac sprue may be due partly to the fact that deficiency of secretin and cholecystokinin may interfere with the action of enterokinase ?
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?

Organism

Organism UniProt Comment Textmining
Homo sapiens
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Posttranslational Modification

Posttranslational Modification Comment Organism
proteolytic modification secreted as a proenzyme, enterokinase converts trypsinogen to trypsin Homo sapiens
side-chain modification glycoprotein Homo sapiens

Source Tissue

Source Tissue Comment Organism Textmining
duodenum mucosa of the first and second portion of the duodenum Homo sapiens
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jejunum jejunal mucosa Homo sapiens
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Substrates and Products (Substrate)

Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
Trypsinogen + H2O enterokinase deficiency is a distinct clinical entity characterized by diarrhea, failure to thrive, hypoproteinemia, and edema. Acquired enterokinase deficiency may occur in some diffuse small bowel diseases. Steatorhea of cellac sprue may be due partly to the fact that deficiency of secretin and cholecystokinin may interfere with the action of enterokinase Homo sapiens ?
-
?