Literature summary for 3.1.1.89 extracted from

Nicholls, R.E.; Sontag, J.M.; Zhang, H.; Staniszewski, A.; Yan, S.; Kim, C.Y.; Yim, M.; Woodruff, C.M.; Arning, E.; Wasek, B.; Yin, D.; Bottiglieri, T.; Sontag, E.; Kandel, E.R.; Arancio, O.
PP2A methylation controls sensitivity and resistance to beta-amyloid-induced cognitive and electrophysiological impairments (2016), Proc. Natl. Acad. Sci. USA, 113, 3347-3352 .

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Organism

Organism	UniProt	Comment	Textmining
Mus musculus	Q8BVQ5	-	-

Synonyms

Synonyms	Comment	Organism
PME-1	-	Mus musculus

General Information

General Information	Comment	Organism
physiological function	generation of transgenic mice that overexpress the PP2A methylesterase, protein phosphatase methylesterase-1 (PME-1). PME-1 overexpression enhances behavioral and electrophysiological impairments caused by exogenous Abeta exposure	Mus musculus

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Release 2023.1 (January 2023)