Cloned (Comment) | Organism |
---|---|
gene NCU00939 | Neurospora crassa |
Organism | UniProt | Comment | Textmining |
---|---|---|---|
Neurospora crassa | Q7SFA8 | - |
- |
Neurospora crassa 74-ORS-6a | Q7SFA8 | - |
- |
Source Tissue | Comment | Organism | Textmining |
---|---|---|---|
mycelium | - |
Neurospora crassa | - |
Synonyms | Comment | Organism |
---|---|---|
DCC-1 protein | - |
Neurospora crassa |
development and carotenogenesis control-1 protein | UniProt | Neurospora crassa |
NCU00939 | - |
Neurospora crassa |
General Information | Comment | Organism |
---|---|---|
malfunction | in contrast to the wild-type strain, which forms mature pigmented perithecia, only an insignificant number of small unpigmented mutant fruiting bodies are observed in the dcc-1 deletion mutant, thus deletion of dcc-1 leads to sterility. Supplementation with cAMP does not restore the sexual defects (formation of submerged perithecia) of the DELTAdcc-1 mutant | Neurospora crassa |
physiological function | two-component signaling pathways are based on phosphoryl group transfer between histidine kinase and response regulator proteins and regulate environmental responses. The DCC-1 protein in Neurospora crasse protein participates in the regulation of processes such as conidiation, perithecial development, and, to a certain degree, carotenogenesis. DCC-1 exerts its effect by promoting cyclic AMP production, including GNA-3 and CR-1, thereby placing this protein within the context of a signaling pathway that operates during conidiation and sexual development. The DCC-1 histidine kinase is required for proper perithecial development, it has a significant role as a negative regulator of sporulation during vegetative growth | Neurospora crassa |