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Literature summary for 2.7.12.1 extracted from

  • Xu, F.; Meng, T.; Li, P.; Yu, Y.; Cui, Y.; Wang, Y.; Gong, Q.; Wang, N.N.
    A soybean dual-specificity kinase, GmSARK, and its Arabidopsis homolog, AtSARK, regulate leaf senescence through synergistic actions of auxin and ethylene (2011), Plant Physiol., 157, 2131-2153.
    View publication on PubMedView publication on EuropePMC

Cloned(Commentary)

Cloned (Comment) Organism
AtSARK, the gene gene consists of 11 exons, quantitative RT-PCR analysis, inducible overexpression of AtSARK in Arabidopsis thaliana causes precocious senescence and abnormal floral organ development Arabidopsis thaliana
GmSARK, quantitative RT-PCR analysis, inducible overexpression in Arabidopsis thaliana leads to early leaf senescence, chloroplast destruction, and abnormal flower morphology. GmSARK-overexpressing seedlings reveal a multitude of changes in phytohormone synthesis and signaling, specifically the repression of cytokinin functions and the induction of auxin and ethylene pathways. Overexpressing GmSARK induces the expression of critical senescence-related transcription factors, including NAC1, NAC2, WRKY6, and AtNAP, in Arabidopsis seedlings Glycine max

Protein Variants

Protein Variants Comment Organism
additional information construction of a a T-DNA insertion mutant of AtSARK that shows significantly delayed senescence Arabidopsis thaliana

Organism

Organism UniProt Comment Textmining
Arabidopsis thaliana
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-
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Glycine max
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-
-

Posttranslational Modification

Posttranslational Modification Comment Organism
phosphoprotein the enzyme autophosphorylates on both serine/threonine and tyrosine residues Glycine max
phosphoprotein the enzyme autophosphorylates on both serine/threonine and tyrosine residues Arabidopsis thaliana

Source Tissue

Source Tissue Comment Organism Textmining
leaf
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Glycine max
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leaf
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Arabidopsis thaliana
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seedling
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Arabidopsis thaliana
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Substrates and Products (Substrate)

Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
additional information the enzyme autophosphorylates on both serine/threonine and tyrosine residues Glycine max ?
-
?
additional information the enzyme autophosphorylates on both serine/threonine and tyrosine residues Arabidopsis thaliana ?
-
?

Synonyms

Synonyms Comment Organism
AtSARK
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Arabidopsis thaliana
GmSARK
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Glycine max
leucine-rich repeat-receptor-like protein kinase
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Glycine max
leucine-rich repeat-receptor-like protein kinase
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Arabidopsis thaliana

General Information

General Information Comment Organism
malfunction a T-DNA insertion mutant of AtSARK shows significantly delayed senescence, while inducible overexpression of AtSARK in Arabidopsis thaliana causes precocious senescence and abnormal floral organ development. phenotypes of transgenic lines, overview Arabidopsis thaliana
malfunction inducible GmSARK overexpression in Arabidopsis thaliana leads to early leaf senescence, chloroplast destruction, and abnormal flower morphology. GmSARK-overexpressing seedlings reveal a multitude of changes in phytohormone synthesis and signaling, specifically the repression of cytokinin functions and the induction of auxin and ethylene pathways. Inhibition of either auxin action or ethylene biosynthesis alleviates the senescence induced by GmSARK. Mutation of either auxin resistant1 or ethylene insensitive2 genes completely reverses the GmSARK-induced senescence. Phenotype, detailed overview Glycine max
metabolism the SARK-mediated pathway may be a widespread mechanism in regulating leaf senescence Glycine max
physiological function enzyme GmSARK mediates leaf senescence, it has specific roles in senescence-inducing hormonal pathways, molecular mechanism, overview Glycine max