Cloned (Comment) | Organism |
---|---|
AtSARK, the gene gene consists of 11 exons, quantitative RT-PCR analysis, inducible overexpression of AtSARK in Arabidopsis thaliana causes precocious senescence and abnormal floral organ development | Arabidopsis thaliana |
GmSARK, quantitative RT-PCR analysis, inducible overexpression in Arabidopsis thaliana leads to early leaf senescence, chloroplast destruction, and abnormal flower morphology. GmSARK-overexpressing seedlings reveal a multitude of changes in phytohormone synthesis and signaling, specifically the repression of cytokinin functions and the induction of auxin and ethylene pathways. Overexpressing GmSARK induces the expression of critical senescence-related transcription factors, including NAC1, NAC2, WRKY6, and AtNAP, in Arabidopsis seedlings | Glycine max |
Protein Variants | Comment | Organism |
---|---|---|
additional information | construction of a a T-DNA insertion mutant of AtSARK that shows significantly delayed senescence | Arabidopsis thaliana |
Organism | UniProt | Comment | Textmining |
---|---|---|---|
Arabidopsis thaliana | - |
- |
- |
Glycine max | - |
- |
- |
Posttranslational Modification | Comment | Organism |
---|---|---|
phosphoprotein | the enzyme autophosphorylates on both serine/threonine and tyrosine residues | Glycine max |
phosphoprotein | the enzyme autophosphorylates on both serine/threonine and tyrosine residues | Arabidopsis thaliana |
Source Tissue | Comment | Organism | Textmining |
---|---|---|---|
leaf | - |
Glycine max | - |
leaf | - |
Arabidopsis thaliana | - |
seedling | - |
Arabidopsis thaliana | - |
Substrates | Comment Substrates | Organism | Products | Comment (Products) | Rev. | Reac. |
---|---|---|---|---|---|---|
additional information | the enzyme autophosphorylates on both serine/threonine and tyrosine residues | Glycine max | ? | - |
? | |
additional information | the enzyme autophosphorylates on both serine/threonine and tyrosine residues | Arabidopsis thaliana | ? | - |
? |
Synonyms | Comment | Organism |
---|---|---|
AtSARK | - |
Arabidopsis thaliana |
GmSARK | - |
Glycine max |
leucine-rich repeat-receptor-like protein kinase | - |
Glycine max |
leucine-rich repeat-receptor-like protein kinase | - |
Arabidopsis thaliana |
General Information | Comment | Organism |
---|---|---|
malfunction | a T-DNA insertion mutant of AtSARK shows significantly delayed senescence, while inducible overexpression of AtSARK in Arabidopsis thaliana causes precocious senescence and abnormal floral organ development. phenotypes of transgenic lines, overview | Arabidopsis thaliana |
malfunction | inducible GmSARK overexpression in Arabidopsis thaliana leads to early leaf senescence, chloroplast destruction, and abnormal flower morphology. GmSARK-overexpressing seedlings reveal a multitude of changes in phytohormone synthesis and signaling, specifically the repression of cytokinin functions and the induction of auxin and ethylene pathways. Inhibition of either auxin action or ethylene biosynthesis alleviates the senescence induced by GmSARK. Mutation of either auxin resistant1 or ethylene insensitive2 genes completely reverses the GmSARK-induced senescence. Phenotype, detailed overview | Glycine max |
metabolism | the SARK-mediated pathway may be a widespread mechanism in regulating leaf senescence | Glycine max |
physiological function | enzyme GmSARK mediates leaf senescence, it has specific roles in senescence-inducing hormonal pathways, molecular mechanism, overview | Glycine max |