Literature summary for 2.7.11.18 extracted from

Moitra, J.; Evenoski, C.; Sammani, S.; Wadgaonkar, R.; Turner, J.R.; Ma, S.F.; Garcia, J.G.
A transgenic mouse with vascular endothelial over-expression of the non-muscle myosin light chain kinase-2 isoform is susceptible to inflammatory lung injury: role of sexual dimorphism and age (2008), Transl. Res., 151, 141-153.

Search for more literature for 2.7.11.18

Protein Variants

Protein Variants	Comment	Organism
additional information	vascular endothelial overexpression of the human nonmuscle isozyme of myosin light chain kinase-2 renders the transgenic mice susceptible to lipopolysaccharide-induced or mechanical-induced inflammatory lung injury, the homozygous mice show reduced fecundity and litter survival until weaning, as well as initially delayed growth compared to wild-type mice, phenotype and modeling, overview	Homo sapiens

Organism

Organism	UniProt	Comment	Textmining
Homo sapiens	-	-	-

Source Tissue

Source Tissue	Comment	Organism	Textmining
additional information	nonmuscle isozyme	Homo sapiens	-

Synonyms

Synonyms	Comment	Organism
myosin light chain kinase	-	Homo sapiens
nmMLCK2	-	Homo sapiens

Use of this online version of BRENDA is free under the CC BY 4.0 license. See terms of use for full details.

Release 2023.1 (January 2023)