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Literature summary for 2.7.10.1 extracted from

  • Miyamoto, T.; Kim, D.; Knox, J.; Johnson, E.; Mucke, L.
    Increasing the Receptor Tyrosine Kinase EphB2 Prevents Amyloid-?-induced Depletion of Cell Surface Glutamate Receptors by a Mechanism That Requires the PDZ-binding Motif of EphB2 and Neuronal Activity. (2016), J. Biol. Chem., 291, 1719-1734.
    View publication on PubMedView publication on EuropePMC

Organism

Organism UniProt Comment Textmining
Mus musculus P54763
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-

Source Tissue

Source Tissue Comment Organism Textmining
neuron
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Mus musculus
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Synonyms

Synonyms Comment Organism
EphB2
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Mus musculus

General Information

General Information Comment Organism
physiological function although receptor tyrosine kinase EphB2 is not directly involved in the amyloid beta-induced depletion of NMDA-type glutamate receptors, increasing its expression may counteract this pathogenic process through a neuronal activity- and PDZ-dependent regulation of AMPA-type glutamate receptors Mus musculus