Literature summary for 2.7.1.107 extracted from

Baum, A.E.; Akula, N.; Cabanero, M.; Cardona, I.; Corona, W.; Klemens, B.; Schulze, T.G.; Cichon, S.; Rietschel, M.; Noethen, M.M.; Georgi, A.; Schumacher, J.; Schwarz, M.; Abou Jamra, R.; Hoefels, S.; Propping, P.; Satagopan, J.; Detera-Wadleigh, S.D.; Hardy, J.; McMahon, F.J.
A genome-wide association study implicates diacylglycerol kinase eta (DGKH) and several other genes in the etiology of bipolar disorder (2008), Mol. Psychiatry, 13, 197-207.

Application

Application	Comment	Organism
medicine	study on genetic basis of bipolar disorder. Of 37 single nucleotide polymorphisms selected for individual genotyping, the strongest association signal is detected at a marker within the diacylglycerol kinase eta. Several genes, each of modest effect, reproducibly influence disease risk. Bipolar disorder may be a polygenic disease	Homo sapiens

Organism	UniProt	Comment	Textmining
Homo sapiens	-	patients with bipolar disorder	-

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Release 2023.1 (January 2023)