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Literature summary for 2.6.1.5 extracted from

  • Nandi, S.S.; Zheng, H.; Sharma, N.M.; Shahshahan, H.R.; Patel, K.P.; Mishra, P.K.
    Lack of miR-133a decreases contractility of diabetic hearts a role for novel cross talk between tyrosine aminotransferase and tyrosine hydroxylase (2016), Diabetes, 65, 3075-3090 .
    View publication on PubMedView publication on EuropePMC
Search for more literature for 2.6.1.5

Organism

Organism UniProt Comment Textmining
Mus musculus Q8QZR1
-
-
Rattus norvegicus P04694
-
-

Source Tissue

Source Tissue Comment Organism Textmining
CATH.a cell
-
 Mus musculus
-
heart
-
 Rattus norvegicus
-
heart
-
 Mus musculus
-
neuron
-
 Rattus norvegicus
-
neuron
-
 Mus musculus
-

Subunits

Subunits Comment Organism
? x * 50000, SDS-PAGE Rattus norvegicus
? x * 50000, SDS-PAGE Mus musculus

Synonyms

Synonyms Comment Organism
TAT
-
 Rattus norvegicus
TAT
-
 Mus musculus
tyrosine aminotransferase
-
 Rattus norvegicus
tyrosine aminotransferase
-
 Mus musculus

Expression

Organism Comment Expression
Mus musculus cardiac-specific miR-133a overexpression prevents upregulation of the enzyme expression in the heart after streptozotocin is administered down
Rattus norvegicus miR-133a mimic treatment improves the contractility of the diabetic rat's heart concomitant with downregulation of the enzyme down

General Information

General Information Comment Organism
metabolism the enzyme inhibits tyrosine hydroxylase in neuronal cells Rattus norvegicus
metabolism the enzyme inhibits tyrosine hydroxylase in neuronal cells Mus musculus