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Literature summary for 2.4.2.30 extracted from

  • Siles, E.; Martinez-Lara, E.; Nunez, M.I.; Munoz-Gamez, J.A.; Martin-Oliva, D.; Valenzuela, M.T.; Peinado, M.A.; Ruiz de Almodovar, J.M.; Javier Oliver, F.
    PARP-1-dependent 3-nitrotyrosine protein modification after DNA damage (2005), J. Cell. Biochem., 96, 709-715.
    View publication on PubMed

Protein Variants

Protein Variants Comment Organism
additional information PARP-1 knockout mice are much less sensitive to inflammatory stress as a result of a diminished release of pro-inflammatory mediators, including nitric oxide, parp-1 knockout mice show reduced NO-induced oxidative injury and response to genotoxic damage during carcinogenesis and inflammation Mus musculus

Natural Substrates/ Products (Substrates)

Natural Substrates Organism Comment (Nat. Sub.) Natural Products Comment (Nat. Pro.) Rev. Reac.
additional information Mus musculus PARP-1 is involved in modulation of NO-derived injury and response to genotoxic damage ?
-
?

Organism

Organism UniProt Comment Textmining
Mus musculus
-
gene parp-1
-

Source Tissue

Source Tissue Comment Organism Textmining
fibroblast
-
Mus musculus
-

Substrates and Products (Substrate)

Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
additional information PARP-1 is involved in modulation of NO-derived injury and response to genotoxic damage Mus musculus ?
-
?

Synonyms

Synonyms Comment Organism
PARP-1
-
Mus musculus

pH Optimum

pH Optimum Minimum pH Optimum Maximum Comment Organism
7.5
-
assay at Mus musculus