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Literature summary for 2.4.1.165 extracted from

  • Martin, P.T.; Xu, R.; Rodino-Klapac, L.R.; Oglesbay, E.; Camboni, M.; Montgomery, C.L.; Shontz, K.; Chicoine, L.G.; Clark, K.R.; Sahenk, Z.; Mendell, J.R.; Janssen, P.M.
    Overexpression of Galgt2 in skeletal muscle prevents injury resulting from eccentric contractions in both mdx and wild-type mice (2009), Am. J. Physiol. Cell Physiol., 296, C476-C488.
    View publication on PubMed
Search for more literature for 2.4.1.165

Application

Application Comment Organism
medicine Galgt2 overexpression inhibits muscular dystrophy in old dystrophic muscles and may have therapeutic impact in many types of muscle injury Mus musculus

Organism

Organism UniProt Comment Textmining
Mus musculus
-
-
-

Source Tissue

Source Tissue Comment Organism Textmining
skeletal muscle
-
 Mus musculus
-

Substrates and Products (Substrate)

Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
UDP-N-acetyl-D-galactosamine + alpha-N-acetylneuraminyl-(2->3)-beta-D-galactosyl-(1->4)-beta-D-glucosyl-(1<->1)-ceramide
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 Mus musculus UDP + N-acetyl-beta-D-galactosaminyl-(1->4)-[alpha-N-acetylneuraminyl-(2->3)]-beta-D-galactosyl-(1->4)-beta-D-glucosyl-(1<->1)-ceramide
-
 ?

Synonyms

Synonyms Comment Organism
CT GalNAc transferase
-
 Mus musculus
cytotoxic T cell GalNAc transferase
-
 Mus musculus
Galgt2
-
 Mus musculus
UDP-GalNAc:beta1,4-N-acetylgalactosaminyltransferase
-
 Mus musculus