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Literature summary for 1.6.3.1 extracted from
- Niacin restriction upregulates NADPH oxidase and reactive oxygen species (ROS) in human keratinocytes (2008), Free Radic. Biol. Med., 44, 527-537.
Application
| Application | Comment | Organism |
|---|---|---|
| medicine | long-term survival mechanisms in niacin deficient HaCaT keratinocyte cells involve accumulation of reactive oxygen species and increased DNA damage with concomitant increase in expression and activity of NAD(P)H oxidase | Homo sapiens |
Organism
| Organism | UniProt | Comment | Textmining |
|---|---|---|---|
| Homo sapiens | - |
- |
- |
Source Tissue
| Source Tissue | Comment | Organism | Textmining |
|---|---|---|---|
| HaCaT cell | keratinocyte cell line. Long-term survival mechanisms in niacin deficient cells involve accumulation of reactive oxygen species and increased DNA damage with concomitant increase in expression and activity of NAD(P)H oxidase | Homo sapiens | - |
| keratinocyte | - |
Homo sapiens | - |
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