1.11.1.20	prostamide/prostaglandin F2alpha synthase	cerebellar cortex	-	707934	
1.4.1.27	glycine cleavage system	cerebellar cortex	in rat brain, the P-protein is confined to astrocytes. The intensity of astrocyte staining varies with regions, with the strongest staining in the hippocampus, the cerebellar cortex, the Bergmann glia in the cerebellum and the Muller cells in the retina	758982	
1.6.5.2	NAD(P)H dehydrogenase (quinone)	cerebellar cortex	the ratio of frontal to cerebellar NQO1 activity is significantly increased in patients with Alzheimer‘s disease versus controls	660005	
2.3.1.B43	protein-lysine desuccinylase (NAD+)	cerebellar cortex	in the cerebellar cortex, SIRT5 can be found in Purkinje cells, and is mainly distributed in the cytoplasm, but is not markedly expressed in the molecular layer or granular layer cells	730103	
2.4.1.255	protein O-GlcNAc transferase	cerebellar cortex	-	658357	
2.4.1.80	ceramide glucosyltransferase	cerebellar cortex	GCS expression is significantly decreased in cortex of Alzheimer disease patients	691551	
2.5.1.16	spermidine synthase	cerebellar cortex	highest levels of expression in nucleus accumbens, taenia tecta, cerebellar cortex, cerebral cortical layer I, hippocampus, hypothalamus, mesencephalic raphe nuclei, central and lateral amygdala, and the circumventricular organs	688510	
2.7.1.158	inositol-pentakisphosphate 2-kinase	cerebellar cortex	-	663251	
2.7.8.1	ethanolaminephosphotransferase	cerebellar cortex	glomeruli	644344	
3.1.4.12	sphingomyelin phosphodiesterase	cerebellar cortex	in hippocampus the enzyme activity is significantly higher than in striatum and cortex. Significant increase in cortical membrane-associated N-Sase activity with age	-, 666446	
3.1.4.54	N-acetylphosphatidylethanolamine-hydrolysing phospholipase D	cerebellar cortex	-	710111	
3.1.6.2	steryl-sulfatase	cerebellar cortex	male, highest enzymic activity in cerebellum followed by cortex and subcortex, female, highest activity in cortex followed by subcortex and cerebellum	666931	
3.4.14.2	dipeptidyl-peptidase II	cerebellar cortex	mRNA expression in ipsilateral and contralateral cortices. Dipeptidyl peptidase II and aminopeptidase N are up-regulated ipsilaterally from 6 h to 7 days post ischemia	732364	
3.4.14.5	dipeptidyl-peptidase IV	cerebellar cortex	mRNA expression in ipsilateral and contralateral cortices. At day 3 post-ischemia, dipeptidyl peptidase IV, 8 and aminopeptidase N are identified in activated microglia and macrophages in the ipsilateral cortex. Seven days post artery occlusion, dipeptidyl peptidase IV immunoreactivity is found in the perikarya of surviving cortical neurons of the ipsilateral hemisphere. At the same time point, dipeptidyl peptidase IV, 8 and aminopeptidase N are targeted in astroglial cells. Total dipeptidyl peptidase IV, 8 and 9 activities remain constant in both hemispheres until day 3 post experimental ischemia, but are increased to 165% in the ipsilateral cortex at day 7	732364	
3.4.14.9	tripeptidyl-peptidase I	cerebellar cortex	-	752408	
3.4.19.6	pyroglutamyl-peptidase II	cerebellar cortex	-	647288	
3.4.21.93	Proprotein convertase 1	cerebellar cortex	ischemic cortex after transient focal cerebral ischemia. Both the mRNA and protein levels of PC1 in ischemic cortices decrease gradually at 4, 8, and 16 hours of reperfusion after 100 min of middle cerebral artery occlusion. After 24 hours of reperfusion, enhanced intensities of signals for PC1 protein are observed, while signals for PC1 mRNA remain low	718167	
3.4.25.1	proteasome endopeptidase complex	cerebellar cortex	-	-, 719539	
4.2.99.18	DNA-(apurinic or apyrimidinic site) lyase	cerebellar cortex	increased nuclear expression of APE1 in neuronal and glial cells in both familial and sporadic Alzheimer	690474	
6.3.1.2	glutamine synthetase	cerebellar cortex	no significant changes in the enzyme activity between morphine-treated and control samples	713737