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Literature summary for 3.5.4.16 extracted from

  • Tie, L.; Li, X.J.; Wang, X.; Channon, K.M.; Chen, A.F.
    Endothelium-specific GTP cyclohydrolase I overexpression accelerates refractory wound healing by suppressing oxidative stress in diabetes (2009), Am. J. Physiol. Endocrinol. Metab., 296, E1423-E1429.
    View publication on PubMedView publication on EuropePMC
Search for more literature for 3.5.4.16

Organism

Organism UniProt Comment Textmining
Mus musculus
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-
-

Source Tissue

Source Tissue Comment Organism Textmining
endothelium
-
 Mus musculus
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Synonyms

Synonyms Comment Organism
GTPCH I
-
 Mus musculus

General Information

General Information Comment Organism
metabolism GTPCH I is the rate-limiting enzyme for de novo tetrahydrobiopterin synthesis Mus musculus
physiological function endothelium-specific GTP cyclohydrolase I overexpression accelerates refractory wound healing in streptozotocin-induced type 1 diabetic mice through enhanced constitutive NOS activity and suppressed oxidative stress Mus musculus