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Literature summary for 3.4.24.86 extracted from

  • Bigdeli, M.R.; Khoshbaten, A.
    In vivo preconditioning with normobaric hyperoxia induces ischemic tolerance partly by triggering tumor necrosis factor-alpha converting enzyme/tumor necrosis factor-alpha/nuclear factor-kappaB (2008), Neuroscience, 153, 671-678.
    View publication on PubMed

Application

Application Comment Organism
medicine in order to explore the role of TACE and NF-kappa B in hyperoxia-induced brain-ischemic tolerance, rodents are exposed to 95% inspired hyperoxia 4 h/day for six consecutive days or for 24 continous hours (prolonged hyperoxia). 24 h after the treatment. TACE, serum TNF-alpha and phosphor-kappaBalpha expression is up-regulated. Brain ischemic tolerance, inspired hyperoxia and prolonged hyperoxia may partly exert their effects via triggering TACE/TNF-alpha /NF-kappaB Rattus norvegicus

Organism

Organism UniProt Comment Textmining
Rattus norvegicus
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Source Tissue

Source Tissue Comment Organism Textmining
brain the expression of TACE is increased in intermittent and prolonged hyperoxia Rattus norvegicus
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Synonyms

Synonyms Comment Organism
TACE
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Rattus norvegicus