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Literature summary for 3.4.24.83 extracted from

  • Zhang, W.W.; Wang, X.; Xie, P.; Yuan, S.T.; Liu, Q.H.
    Anthrax lethal toxin suppresses high glucose induced VEGF over secretion through a post-translational mechanism (2015), Int. J. Ophthalmol., 8, 453-458 .
    View publication on PubMedView publication on EuropePMC
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Metals/Ions

Metals/Ions Comment Organism Structure
Zn2+ zinc metalloproteinase Bacillus anthracis

Natural Substrates/ Products (Substrates)

Natural Substrates Organism Comment (Nat. Sub.) Natural Products Comment (Nat. Pro.) Rev. Reac.
mitogen-activated protein kinase kinase 1 + H2O Bacillus anthracis
-
 ?
-
 ?

Organism

Organism UniProt Comment Textmining
Bacillus anthracis P15917
-
-

Source Tissue

Source Tissue Comment Organism Textmining

Substrates and Products (Substrate)

Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
mitogen-activated protein kinase kinase 1 + H2O
-
 Bacillus anthracis ?
-
 ?

Synonyms

Synonyms Comment Organism
anthrax lethal toxin
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 Bacillus anthracis
LeTx
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 Bacillus anthracis

General Information

General Information Comment Organism
physiological function high glucose-induced activation of the MAPK extracellular signal-regulated kinase (ERK1/2) and p38 in the ARPE cell line is blocked by the enzyme. High glucose induced ARPE cells over proliferation is inhibited by the enzyme Bacillus anthracis