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Literature summary for 2.4.1.38 extracted from

  • Yang, H.; Hu, L.; Chen, J.; Zhu, J.; Tao, T.; Zhang, F.; Li, X.; He, X.; Shen, A.; Cheng, C.
    Lipopolysaccharide induced upregulation of beta-1,4-galactosyltransferase-I in Schwann cell (2009), Inflammation, 32, 279-286.
    View publication on PubMed

Localization

Localization Comment Organism GeneOntology No. Textmining
Golgi apparatus
-
Rattus norvegicus 5794
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membrane
-
Rattus norvegicus 16020
-

Organism

Organism UniProt Comment Textmining
Rattus norvegicus
-
-
-

Source Tissue

Source Tissue Comment Organism Textmining
RSC96 cell
-
Rattus norvegicus
-
Schwann cell
-
Rattus norvegicus
-

Substrates and Products (Substrate)

Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
additional information beta-1,4-GalT-I transfers galactose from UDP-galactose donors to terminal N-acetylglucosamine of carbohydrate chains in beta-1,4-linkage to form a Galbeta1->4GlcNAc structure Rattus norvegicus ?
-
?

Synonyms

Synonyms Comment Organism
beta-1,4-galactosyltransferase-I
-
Rattus norvegicus
beta-1,4-GalT-I
-
Rattus norvegicus

Expression

Organism Comment Expression
Rattus norvegicus beta-1,4-GalT-I mRNA is significant increased 2 h after lipopolysaccharide stimulation with 0.001 mg/ml up

General Information

General Information Comment Organism
physiological function beta-1,4-GalT-I may play an important role in adhesion and migration of Schwann cells during inflammation Rattus norvegicus