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Literature summary for 2.3.2.13 extracted from

  • Nahrendorf, M.; Aikawa, E.; Figueiredo, J.L.; Stangenberg, L.; van den Borne, S.W.; Blankesteijn, W.M.; Sosnovik, D.E.; Jaffer, F.A.; Tung, C.H.; Weissleder, R.
    Transglutaminase activity in acute infarcts predicts healing outcome and left ventricular remodelling: implications for FXIII therapy and antithrombin use in myocardial infarction (2008), Eur. Heart J., 29, 445-454.
    View publication on PubMed

Application

Application Comment Organism
medicine patients with low FXIII plasma levels, which occur naturally after myocardial infarction, may potentially benefit from transient and local augmentation of FXIII activity in the infarct Homo sapiens

Organism

Organism UniProt Comment Textmining
Homo sapiens
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-
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Mus musculus
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-
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Source Tissue

Source Tissue Comment Organism Textmining
myocardium in the mouse model of coronary ligation, modulation of FXIII activity by therapy impacts myocardial healing Mus musculus
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myocardium transglutaminase factor XIII levels are diminished in patients after infarct rupture. Transglutaminase factor XIII activity peaks on day 3 after myocardial infarction Homo sapiens
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Synonyms

Synonyms Comment Organism
factor XIII
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Homo sapiens
transglutaminase factor XIII
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Mus musculus
transglutaminase factor XIII
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Homo sapiens