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BRENDA support

Literature summary for 1.4.3.4 extracted from

  • Baler, R.D.; Volkow, N.D.; Fowler, J.S.; Benveniste, H.
    Is fetal brain monoamine oxidase inhibition the missing link between maternal smoking and conduct disorders? (2008), J. Psychiatry Neurosci., 33, 187-195.
    View publication on PubMedView publication on EuropePMC

Application

Application Comment Organism
medicine theoretical and experimental support for the notion that cigarette smoke–induced inhibition of MAO in the fetal brain, particularly when it occurs in combination with polymorphisms in the MAOA gene that lead to lower enzyme concentration in the brain, may result in brain morphologic and functional changes that enhance the risk of irritability, poor self-control and aggression in the offspring Homo sapiens

Organism

Organism UniProt Comment Textmining
Homo sapiens P21397
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Source Tissue

Source Tissue Comment Organism Textmining
brain theoretical and experimental support for the notion that cigarette smoke-induced inhibition of MAO in the fetal brain, particularly when it occurs in combination with polymorphisms in the MAOA gene that lead to lower enzyme concentration in the brain, may result in brain morphologic and functional changes that enhance the risk of irritability, poor self-control and aggression in the offspring Homo sapiens
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